Cerebral cortical phospholipase A2 activity of senescence-accelerated mouse is increased in an age-dependent manner

Neurosci Res. 1997 Nov;29(3):269-72. doi: 10.1016/s0168-0102(97)00098-9.

Abstract

The PLA2 activities of both the cytosolic and the membrane fractions from the cerebral cortex of 17-month old senescence-accelerated mouse-prone/10 (SAM-P/10) were significantly increased by 1.3 times compared with those of 2-month old mice. The PLA2 activities were independent of Ca2+ ion and not affected either by dithiothreitol (DTT) or by trifluoromethyl ketone analogs of arachidonic acid (AACOCF3). The PLA2 activities were eluted in a single peak of molecular mass of 170 kDa, using a gel filtration column. These findings suggest that the enhanced PLA2 activity may be related to neuronal degeneration and accelerated senescence of SAM-P/10, and cerebral cortical PLA2 activity categorized as a Ca2+-independent PLA2.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aging / genetics
  • Aging / physiology*
  • Animals
  • Arachidonic Acids / pharmacology
  • Calcium / pharmacology
  • Cerebral Cortex / enzymology*
  • Cerebral Cortex / growth & development
  • Dithiothreitol / pharmacology
  • Male
  • Mice
  • Mice, Inbred Strains
  • Nerve Degeneration
  • Phospholipases A / metabolism*
  • Phospholipases A2

Substances

  • Arachidonic Acids
  • Phospholipases A
  • Phospholipases A2
  • Calcium
  • Dithiothreitol