Pharmacokinetics of the multidrug-resistance-converting drug dexniguldipine and its pyridine metabolite M-1 in the plasma, tumor, and renal tissue of tumor-bearing Wag/Rij rats

Cancer Chemother Pharmacol. 1997;41(1):48-52. doi: 10.1007/s002800050706.

Abstract

The pharmacokinetics of oral dexniguldipine, a new multidrug-resistance-modifying agent under clinical evaluation, and its pyridine metabolite M-1 were determined in plasma, tumor, and renal tissue in Wag/Rij rats bearing a multidrug-resistant CC531 colon adenocarcinoma tumor under the renal capsule. The pharmacokinetics were studied in four experiments. After a single administration of dexniguldipine (30 mg/kg), tumors and kidneys were collected after 5 (experiment 1), 24 (experiment 2), and 48 h (experiment 3). In the fourth experiment, dexniguldipine was given once daily for 3 consecutive days at a dose of 30 mg/kg. In all experiments, plasma samples were collected at regular intervals. The concentrations of dexniguldipine and M-1 could be determined in plasma in most of the rats at up to 32 h after drug administration. The area under the curve (AUC) of dexniguldipine and M-1 varied by a factor of 2-6 in the four experiments. High tumor-tissue concentrations of dexniguldipine were observed. The concentrations were highest in the multiple-dose experiment (2014 +/- 1005 ng/g tissue). High degrees of correlation (> 0.8) were established between the concentrations of dexniguldipine measured in plasma and tumor as well as renal tissue. Overall, tumor-tissue concentrations of M-1 comprised one-third of the dexniguldipine concentrations measured.

MeSH terms

  • Adenocarcinoma / blood
  • Adenocarcinoma / metabolism*
  • Adenocarcinoma / pathology
  • Animals
  • Antineoplastic Agents / administration & dosage
  • Antineoplastic Agents / pharmacokinetics*
  • Colonic Neoplasms / blood
  • Colonic Neoplasms / metabolism*
  • Colonic Neoplasms / pathology
  • Dihydropyridines / administration & dosage
  • Dihydropyridines / pharmacokinetics*
  • Drug Resistance, Multiple
  • Drug Resistance, Neoplasm
  • Humans
  • Kidney / metabolism*
  • Neoplasm Transplantation
  • Pyridines / metabolism
  • Rats
  • Rats, Inbred Strains

Substances

  • Antineoplastic Agents
  • Dihydropyridines
  • Pyridines
  • niguldipine