Hindlimb unloading in rat decreases preosteoblast proliferation assessed in vivo with BrdU incorporation

Am J Physiol. 1998 Jan;274(1):E108-14. doi: 10.1152/ajpendo.1998.274.1.E108.

Abstract

Immobilization affects bone formation. However, the mechanisms regulating the decrease in osteoblast recruitment remain unclear. The aim of our study was to determine in vivo osteoblastic proliferation after short-term immobilization among the different bone compartments. Twelve Wistar 5-wk-old rats were assigned to two groups: six tail-suspended animals for 6 days and their six age-related controls. Osmotic minipumps, each containing 40 mg of bromodeoxyuridine (BrdU), were implanted intraperitoneally at day 4 until euthanasia. Histomorphometric measurements found a significantly lower bone volume in primary (ISP, -22%) and secondary spongiosa (IISP, -37%) in unloaded rats compared with their age-related controls. BrdU immunohistochemistry showed that the proliferation capacity of osteogenic precursors in ISP (-29%) and preosteoblasts in IISP (-80%) and in periosteum as well as bone marrow cells (-40%) was lowered by unloading. We demonstrated in vivo for the first time that 6-day tail suspension induced a significant decrease in proliferation of periosteal and trabecular preosteoblasts in ISP and IISP as well as in bone marrow cells.

MeSH terms

  • Animals
  • Bone Marrow Cells / cytology
  • Bone and Bones / cytology
  • Bromodeoxyuridine
  • Cell Division
  • Hindlimb Suspension / physiology*
  • Male
  • Osteoblasts / cytology*
  • Osteogenesis / physiology*
  • Rats
  • Rats, Wistar

Substances

  • Bromodeoxyuridine