Overexpression of bcl-2 protein inhibits terminal differentiation of oral keratinocytes in vitro

J Oral Pathol Med. 1998 Jan;27(1):11-7. doi: 10.1111/j.1600-0714.1998.tb02084.x.

Abstract

The bcl-2 proto-oncogene is a known inhibitor of apoptosis; in normal human stratified squamous epithelium, its expression is restricted to the basal cell layer. To investigate the functional role of bcl-2 protein in the process of differentiation of oral keratinocytes, bcl-2 expression vector was transfected into SCC-25 cells, which normally undergo squamous cell differentiation in vitro while expressing specific differentiation markers, e.g., keratin 10/11 and involucrin. In bcl-2 transfected SCC-25 cells, the expression of these differentiation markers was markedly suppressed. The bcl-2 proto-oncogene may play a critical role in opposing the commitment to terminal differentiation and apoptosis of oral keratinocytes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / genetics*
  • Apoptosis / physiology
  • Carcinoma, Squamous Cell / genetics*
  • Cell Differentiation / physiology
  • DNA Fragmentation
  • Down-Regulation
  • Genes, bcl-2
  • Humans
  • Immunoenzyme Techniques
  • Keratinocytes / metabolism*
  • Keratins / biosynthesis
  • Mouth Neoplasms / genetics*
  • Protein Precursors / biosynthesis
  • Proto-Oncogene Mas
  • Proto-Oncogene Proteins c-bcl-2 / biosynthesis
  • Proto-Oncogene Proteins c-bcl-2 / physiology*
  • Tumor Cells, Cultured

Substances

  • MAS1 protein, human
  • Protein Precursors
  • Proto-Oncogene Mas
  • Proto-Oncogene Proteins c-bcl-2
  • involucrin
  • Keratins