Effects of a urinary trypsin inhibitor on acute circulatory insufficiency after surgical operation

Am J Surg. 1998 Feb;175(2):142-5. doi: 10.1016/s0002-9610(97)00263-8.

Abstract

Objective: To assess the effectiveness of an urinary trypsin inhibitor (UTI) on a surgical stress, particularly the influences on cytokines and diuretic hormones.

Subjects and methods: Sixteen patients with carcinoma of the digestive system and predicted to suffer from circulatory insufficiency were enrolled. Selection of group was divided alternatively. UTI was administered for 5 consecutive days, at a dose of 300,000 units per day. Urine and blood specimens were collected before, immediately after, and 1, 3, and 5 days after surgery. Interleukin 8 (IL-8), polymorphonuclear leukocyte elastase (PMNE), vasopressin (ADH), atrial natriuretic peptide (ANP), angiotensin II (AT-II), and endothelin 1 (ET-1) in the blood, and N-acetyl-D-glucosaminidase (NAG) in the urine, were determined.

Results: A UTI group was 9 patients, and a control group was 7 patients. The operation time was significantly longer in the UTI group than in the control group. In the UTI group, the elevation of IL-8, PMNE/WBC, ADH, urinary NAG, and BUN were significantly inhibited. AT-II and ET-1, in the UTI group, tended to be suppressed, and ANP showed the similar changes in the two groups.

Conclusion: UTI is considered effective in the prevention of excessive reaction against major surgery.

Publication types

  • Clinical Trial
  • Randomized Controlled Trial

MeSH terms

  • Angiotensin II / blood
  • Angiotensin II / urine
  • Atrial Natriuretic Factor / blood
  • Atrial Natriuretic Factor / urine
  • Blood Circulation / drug effects
  • Digestive System Neoplasms / physiopathology*
  • Digestive System Neoplasms / surgery
  • Female
  • Glycoproteins / pharmacology*
  • Humans
  • Interleukin-8 / blood
  • Interleukin-8 / urine
  • Leukocyte Elastase / blood
  • Leukocyte Elastase / urine
  • Male
  • Middle Aged
  • Postoperative Period
  • Stress, Physiological / metabolism*
  • Trypsin Inhibitors / pharmacology*

Substances

  • Glycoproteins
  • Interleukin-8
  • Trypsin Inhibitors
  • Angiotensin II
  • Atrial Natriuretic Factor
  • Leukocyte Elastase
  • urinastatin