Bacterial expression and characterization of human secretory class V phospholipase A2

Biochem J. 1998 Apr 15;331 ( Pt 2)(Pt 2):353-7. doi: 10.1042/bj3310353.

Abstract

Mammalian secretory class V phospholipase A2 (PLA2) is a newly discovered PLA2 that is implicated in eicosanoid formation in inflammatory cells. As a first step towards understanding the structure, function and regulation of this PLA2, we constructed a bacterial expression vector for human secretory class V PLA2 (hV-PLA2), over-expressed and purified the protein, and determined its physical and kinetic properties. When compared with human class IIa enzyme (hIIa-PLA2), hV-PLA2 has several distinct properties. First, hV-PLA2 can catalyse the hydrolysis of phosphatidylcholine more effectively than hIIa-PLA2 by two orders of magnitude. Secondly, hV-PLA2 has much higher binding affinity and activity for compactly packed phosphatidylcholine bilayers than hIIa-PLA2. Finally, hV-PLA2 has much reduced thermal stability compared with hIIa-PLA2. These data suggest that hV-PLA2 is better suited than hIIa-PLA2 for acting on the outer cellular membrane and liberating arachidonic acid from membrane phospholipids. Also, the unusually low thermal stability of hV-PLA2 might contribute to tighter regulation of its activities in extracellular media.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Amino Acid Sequence
  • Base Sequence
  • Cell Membrane / metabolism
  • Enzyme Activation
  • Enzyme Stability
  • Escherichia coli / genetics*
  • Gene Expression*
  • Hot Temperature
  • Humans
  • Hydrolysis
  • Kinetics
  • Liposomes / metabolism
  • Molecular Sequence Data
  • Phospholipases A / chemistry
  • Phospholipases A / genetics*
  • Phospholipases A / metabolism
  • Phospholipases A2
  • Phospholipids / metabolism
  • Polymers
  • Protein Folding
  • Recombinant Proteins
  • Substrate Specificity

Substances

  • Liposomes
  • Phospholipids
  • Polymers
  • Recombinant Proteins
  • Phospholipases A
  • Phospholipases A2