Neurotrophins differentially regulate voltage-gated ion channels

Mol Cell Neurosci. 1997;10(3-4):173-83. doi: 10.1006/mcne.1997.0656.

Abstract

Neurotrophic factors profoundly affect neuronal differentiation, but whether they influence neuronal phenotype in instructive ways remains unclear: do different neurotrophic factors always trigger identical programs of differentiation or can each impose distinct functional properties even when acting upon the same population of target neurons? We addressed this issue by examining the regulatory effects of the four neurotrophins on the molecular components of electrical excitability, voltage-gated ion channels, within a single cellular context. Using patch clamp methods, we studied neurotrophin regulation of voltage-gated sodium, calcium, and potassium currents in SK-N-SH neuroblastoma cells. We found that each neurotrophin induced a unique pattern of expression of ionic currents despite similar activation of initial signal transduction events. Thus, each neurotrophin imposed a different excitable phenotype even when acting upon the same target cells.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Calcium Channels / drug effects
  • Calcium Channels / metabolism
  • GAP-43 Protein / drug effects
  • GAP-43 Protein / metabolism
  • Humans
  • Ion Channel Gating / drug effects
  • Ion Channel Gating / physiology*
  • Ion Channels / drug effects
  • Ion Channels / metabolism*
  • Nerve Growth Factors / physiology*
  • Neuroblastoma
  • Potassium Channels / drug effects
  • Potassium Channels / metabolism
  • Signal Transduction / drug effects
  • Sodium Channels / drug effects
  • Sodium Channels / metabolism
  • Tumor Cells, Cultured

Substances

  • Calcium Channels
  • GAP-43 Protein
  • Ion Channels
  • Nerve Growth Factors
  • Potassium Channels
  • Sodium Channels