[Immunology in clinical practice. VIII. Role of cytokines in the pathogenesis of bacterial infections]

Ned Tijdschr Geneeskd. 1998 Jan 3;142(1):14-7.
[Article in Dutch]

Abstract

Cytokines are small proteins that can be produced by many different cell types involved in inflammation. The cytokine network consists of pro-inflammatory cytokines, facilitating inflammation, and anti-inflammatory cytokines, reducing inflammation at least in part by inhibiting the production of pro-inflammatory cytokines. The production of cytokines is enhanced during bacterial infections. The balance between pro- and anti-inflammatory cytokines critically determines the outcome of infection. Excessive systemic release of pro-inflammatory cytokines such as tumour necrosis factor alpha and interleukin 1 may contribute to organ failure in patients with fulminant sepsis. On the other hand, the local action of pro-inflammatory cytokines, at the site of a compartmentalized infection, is required for an adequate host antibacterial defence. Therefore, neutralization of pro-inflammatory cytokines may benefit some patients with bacterial sepsis but harm others.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Bacterial Infections / complications
  • Bacterial Infections / immunology*
  • Cytokines / biosynthesis
  • Cytokines / immunology*
  • Humans
  • Interleukin-1 / biosynthesis
  • Interleukin-10 / immunology
  • Multiple Organ Failure / etiology
  • Receptors, Interleukin-1
  • Sepsis / immunology
  • Tumor Necrosis Factor-alpha / biosynthesis

Substances

  • Cytokines
  • Interleukin-1
  • Receptors, Interleukin-1
  • Tumor Necrosis Factor-alpha
  • Interleukin-10