Insulin down-regulates angiotensinogen gene expression and angiotensinogen secretion in cultured adipose cells

Biochem Biophys Res Commun. 1998 Sep 8;250(1):77-82. doi: 10.1006/bbrc.1998.9185.

Abstract

Adipose tissue is an important source of angiotensinogen (AT) after liver. Since an association exists between body mass index, hypertension, and insulin-resistance, the role of insulin on the regulation of AT gene expression and AT secretion was examined in cultured Ob1771 and 3T3-F442A adipose cells. Within a physiological range of concentrations (1-17 nM), insulin exerted a negative effect on the abundance of AT mRNA and the secretion of AT. Alterations of insulin-resistance by treatment of adipose cells with TNF-alpha or the thiazolidinedione BRL49653 led respectively to a decrease or an increase in the potency of insulin to down-regulate AT gene expression, whereas maximal inhibition by insulin increased from 30% in TNFalpha-treated cells to 60% in BRL49653-treated cells. These results suggest that a potential link between insulin resistance and high blood pressure may exist by means of increased AT secretion from adipose tissue, especially in obese subjects.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3T3 Cells
  • Adipose Tissue / drug effects*
  • Adipose Tissue / metabolism
  • Angiotensinogen / genetics*
  • Angiotensinogen / metabolism
  • Animals
  • Cells, Cultured
  • Down-Regulation / drug effects*
  • Gene Expression Regulation / drug effects*
  • Hypoglycemic Agents / pharmacology
  • Insulin / pharmacology*
  • Mice
  • RNA, Messenger / genetics
  • Rosiglitazone
  • Thiazoles / pharmacology
  • Thiazolidinediones*
  • Tumor Necrosis Factor-alpha / pharmacology

Substances

  • Hypoglycemic Agents
  • Insulin
  • RNA, Messenger
  • Thiazoles
  • Thiazolidinediones
  • Tumor Necrosis Factor-alpha
  • Rosiglitazone
  • Angiotensinogen