Cyclic AMP elevation is sufficient to promote the survival of spinal motor neurons in vitro

J Neurosci. 1998 Sep 15;18(18):7361-71. doi: 10.1523/JNEUROSCI.18-18-07361.1998.

Abstract

The short-term survival of highly purified embryonic spinal motor neurons (SMNs) in culture can be promoted by many peptide trophic factors, including brain-derived neurotrophic factor (BDNF), ciliary neurotrophic factor (CNTF), fibroblast growth factor (FGF), glial-derived neurotrophic factor (GDNF), and hepatocyte growth factor (HGF). We have asked whether these peptides are sufficient to promote the long-term survival of purified E15 SMNs. Contrary to previous reports, we find that when SMNs are cultured in serum-free medium containing a single peptide trophic factor only approximately one-third of the cells survive for 3 d in culture. When multiple factors are combined, additive effects on survival are observed transiently, but by 7 d of culture the majority of SMNs has died. Surprisingly, when cAMP levels are elevated, the majority of SMNs extend processes and survive for 1 week in culture in the absence of peptide trophic factors, even in low-density cultures. A combination of five peptide trophic factors, together with cAMP elevation, promotes the long-term survival of most of the SMNs in serum-free culture for 3 weeks. These findings provide useful culture conditions for studying the properties of SMNs and have implications for the treatment of motor neuron diseases.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • 1-Methyl-3-isobutylxanthine / pharmacology
  • Animals
  • Bone Morphogenetic Proteins / metabolism
  • Brain-Derived Neurotrophic Factor / pharmacology
  • Cell Survival / physiology
  • Ciliary Neurotrophic Factor
  • Colforsin / pharmacology
  • Cyclic AMP / metabolism*
  • Estradiol / pharmacology
  • Glial Cell Line-Derived Neurotrophic Factor
  • Hepatocyte Growth Factor / pharmacology
  • Hydrocortisone / pharmacology
  • Hypoglycemic Agents / pharmacology
  • Insulin / pharmacology
  • Motor Neurons / cytology*
  • Motor Neurons / drug effects
  • Motor Neurons / metabolism*
  • Nerve Degeneration / metabolism
  • Nerve Growth Factors*
  • Nerve Tissue Proteins / pharmacology
  • Oxygen / pharmacology
  • Phosphodiesterase Inhibitors / pharmacology
  • Progesterone / pharmacology
  • Rats
  • Rats, Sprague-Dawley
  • Signal Transduction / drug effects
  • Signal Transduction / physiology
  • Spinal Cord / cytology*
  • Thyroxine / pharmacology
  • Tretinoin / pharmacology

Substances

  • Bone Morphogenetic Proteins
  • Brain-Derived Neurotrophic Factor
  • Ciliary Neurotrophic Factor
  • Gdnf protein, rat
  • Glial Cell Line-Derived Neurotrophic Factor
  • Hypoglycemic Agents
  • Insulin
  • Nerve Growth Factors
  • Nerve Tissue Proteins
  • Phosphodiesterase Inhibitors
  • Colforsin
  • Progesterone
  • Estradiol
  • Tretinoin
  • Hepatocyte Growth Factor
  • Cyclic AMP
  • Thyroxine
  • Oxygen
  • 1-Methyl-3-isobutylxanthine
  • Hydrocortisone