Serotonin and cholecystokinin activate different populations of rat mesenteric vagal afferents

Neurosci Lett. 1998 Oct 16;255(2):63-6. doi: 10.1016/s0304-3940(98)00690-9.

Abstract

This electrophysiological study was performed to elucidate the interactions of serotonin (5-hydroxytryptamine, 5-HT) and cholecystokinin (CCK) on mesenteric afferents supplying the rat jejunum. 5-HT and CCK produced characteristic responses in multi-unit recordings of mesenteric afferents. Waveform analysis to extract single units from the whole nerve recording identified populations of single afferents that were sensitive to either 5-HT or CCK, but not both. Furthermore, devazepide (0.5 mg/kg) completely abolished the response to CCK without altering the response to 5-HT while granisetron (0.5 mg/kg) abolished the response to 5-HT with no effect on the response to CCK. These results suggest that there are discrete, noninteractive populations of jejunal afferents that possess either 5-HT3 or CCK-A receptors but not both.

MeSH terms

  • Animals
  • Cholecystokinin / antagonists & inhibitors
  • Cholecystokinin / pharmacology*
  • Devazepide / pharmacology
  • Electrophysiology
  • Granisetron / pharmacology
  • Hormone Antagonists / pharmacology
  • Jejunum / innervation*
  • Male
  • Neurons, Afferent / drug effects*
  • Neurons, Afferent / physiology
  • Rats
  • Rats, Wistar
  • Serotonin / pharmacology*
  • Serotonin Antagonists / pharmacology
  • Vagus Nerve / cytology
  • Vagus Nerve / drug effects*

Substances

  • Hormone Antagonists
  • Serotonin Antagonists
  • Serotonin
  • Cholecystokinin
  • Devazepide
  • Granisetron