Normal epithelial cells modulating HLA class I surface molecules are susceptible to lysis mediated by CD3(+) and CD3(-) "nonspecific" killer cells

Cell Immunol. 1998 Dec 15;190(2):183-90. doi: 10.1006/cimm.1998.1392.

Abstract

Natural killer (NK) cells and NK-like T cells have been described as efficiently lysing neoplastic cells derived from hematopoietic tumors. By modulating the expression of HLA class I surface molecules on normal epithelial cells, we also observed that nonneoplastic cells can efficiently be lysed by "nonspecific" effectors. Clonal analysis clearly demonstrates that the presence of HLA-specific inhibitory NK receptors, such as CD94, CD158a, and CD158b, described on NK cells, is responsible for the inhibitory signal. Thus, NK cells, as well as NK-like T cells, in the absence of HLA surface molecules on normal target cells, efficiently lyse epithelial cells.

MeSH terms

  • Antibodies, Monoclonal / immunology
  • CD3 Complex / immunology*
  • Cell Membrane / metabolism
  • Cells, Cultured
  • Cytotoxicity Tests, Immunologic
  • Epithelial Cells / immunology*
  • Female
  • Histocompatibility Antigens Class I / biosynthesis
  • Histocompatibility Antigens Class I / immunology*
  • Humans
  • Killer Cells, Natural / immunology*
  • Leukocytes, Mononuclear / immunology

Substances

  • Antibodies, Monoclonal
  • CD3 Complex
  • Histocompatibility Antigens Class I