IL-4 enhances IL-10 gene expression in murine Th2 cells in the absence of TCR engagement

J Immunol. 1999 Jan 1;162(1):238-44.

Abstract

Both IL-4 and IL-10 are regulatory cytokines produced by Th2 cells that can down-regulate cell-mediated immune responses. The studies reported here examine the influence of various cytokines in the regulation of T cell IL-10 production. The results indicate that IL-10 gene expression by TCR transgenic Th2 cells is significantly up-regulated by IL-4 in the absence of TCR signals. IL-4 enhances both IL-10 mRNA levels and secreted protein, and this effect is not related to enhanced mRNA stability. TCR-mediated IL-10 gene expression is inhibited by cyclosporin A, but IL-4-mediated IL-10 expression is not. IL-4 also enhances IL-13 mRNA levels, to a lesser extent than IL-10, but does not significantly effect the expression of other cytokine mRNAs. Furthermore, IL-4 does not significantly enhance IL-10 expression in Th1 cells. IL-2 also enhances effector cytokine production in the absence of TCR signals, but in a subset nonspecific manner, increasing both Th2 IL-4 mRNA and Th1 IFN-gamma mRNA. These data suggest that Th2 IL-4 production may contribute to the down-regulation of immune responses by directly enhancing Th2 IL-10 production. In addition, the data clearly demonstrate that exogenous cytokines can significantly influence effector cytokine production by effector T cells without the requirement for TCR signals.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adjuvants, Immunologic / physiology*
  • Animals
  • Cells, Cultured
  • Cyclosporine / pharmacology
  • Gene Expression Regulation / drug effects
  • Gene Expression Regulation / immunology*
  • Interleukin-10 / biosynthesis
  • Interleukin-10 / genetics*
  • Interleukin-2 / physiology
  • Interleukin-4 / physiology*
  • Janus Kinase 2
  • Mice
  • Mice, Inbred AKR
  • Mice, Inbred BALB C
  • Mice, Transgenic
  • Protein-Tyrosine Kinases / antagonists & inhibitors
  • Proto-Oncogene Proteins*
  • RNA, Messenger / biosynthesis
  • RNA, Messenger / drug effects
  • RNA, Messenger / metabolism
  • Receptors, Antigen, T-Cell / metabolism*
  • Th1 Cells / enzymology
  • Th1 Cells / metabolism
  • Th2 Cells / enzymology
  • Th2 Cells / metabolism*
  • Tyrphostins / pharmacology
  • Up-Regulation / drug effects
  • Up-Regulation / genetics
  • Up-Regulation / immunology

Substances

  • Adjuvants, Immunologic
  • Interleukin-2
  • Proto-Oncogene Proteins
  • RNA, Messenger
  • Receptors, Antigen, T-Cell
  • Tyrphostins
  • alpha-cyano-(3,4-dihydroxy)-N-benzylcinnamide
  • Interleukin-10
  • Interleukin-4
  • Cyclosporine
  • Protein-Tyrosine Kinases
  • Jak2 protein, mouse
  • Janus Kinase 2