Di Matola T - Search Results

1

Iodide excess induces apoptosis in thyroid cells through a p53-independent mechanism involving oxidative stress.

Vitale M, Di Matola T, D'Ascoli F, Salzano S, Bogazzi F, Fenzi G, Martino E, Rossi G. Vitale M, et al. Among authors: di matola t. Endocrinology. 2000 Feb;141(2):598-605. doi: 10.1210/endo.141.2.7291. Endocrinology. 2000. PMID: 10650940

2

Integrin-dependent cell growth and survival are mediated by different signals in thyroid cells.

Illario M, Amideo V, Casamassima A, Andreucci M, di Matola T, Miele C, Rossi G, Fenzi G, Vitale M. Illario M, et al. Among authors: di matola t. J Clin Endocrinol Metab. 2003 Jan;88(1):260-9. doi: 10.1210/jc.2002-020774. J Clin Endocrinol Metab. 2003. PMID: 12519863

3

Integrin binding to immobilized collagen and fibronectin stimulates the proliferation of human thyroid cells in culture.

Vitale M, Illario M, Di Matola T, Casamassima A, Fenzi G, Rossi G. Vitale M, et al. Among authors: di matola t. Endocrinology. 1997 Apr;138(4):1642-8. doi: 10.1210/endo.138.4.5052. Endocrinology. 1997. PMID: 9075726

4

Fibronectin is required to prevent thyroid cell apoptosis through an integrin-mediated adhesion mechanism.

Vitale M, Di Matola T, Fenzi G, Illario M, Rossi G. Vitale M, et al. Among authors: di matola t. J Clin Endocrinol Metab. 1998 Oct;83(10):3673-80. doi: 10.1210/jcem.83.10.5175. J Clin Endocrinol Metab. 1998. PMID: 9768683

5

Prenyltransferase inhibitors induce apoptosis in proliferating thyroid cells through a p53-independent CrmA-sensitive, and caspase-3-like protease-dependent mechanism.

Vitale M, Di Matola T, Rossi G, Laezza C, Fenzi G, Bifulco M. Vitale M, et al. Among authors: di matola t. Endocrinology. 1999 Feb;140(2):698-704. doi: 10.1210/endo.140.2.6494. Endocrinology. 1999. PMID: 9927296

6

Apoptosis induced by denied adhesion to extracellular matrix (anoikis) in thyroid epithelial cells is p53 dependent but fails to correlate with modulation of p53 expression.

Vitale M, Di Matola T, Bifulco M, Casamassima A, Fenzi G, Rossi G. Vitale M, et al. Among authors: di matola t. FEBS Lett. 1999 Nov 26;462(1-2):57-60. doi: 10.1016/s0014-5793(99)01512-4. FEBS Lett. 1999. PMID: 10580091 Free article.

7

Serum withdrawal-induced apoptosis in thyroid cells is caused by loss of fibronectin-integrin interaction.

Di Matola T, Mueller F, Fenzi G, Rossi G, Bifulco M, Marzano LA, Vitale M. Di Matola T, et al. J Clin Endocrinol Metab. 2000 Mar;85(3):1188-93. doi: 10.1210/jcem.85.3.6425. J Clin Endocrinol Metab. 2000. PMID: 10720060

8

Amiodarone induces cytochrome c release and apoptosis through an iodine-independent mechanism.

Di Matola T, D'Ascoli F, Fenzi G, Rossi G, Martino E, Bogazzi F, Vitale M. Di Matola T, et al. J Clin Endocrinol Metab. 2000 Nov;85(11):4323-30. doi: 10.1210/jcem.85.11.6995. J Clin Endocrinol Metab. 2000. PMID: 11095475

9

Lovastatin-induced apoptosis in thyroid cells: involvement of cytochrome c and lamin B.

Di Matola T, D'Ascoli F, Luongo C, Bifulco M, Rossi G, Fenzi G, Vitale M. Di Matola T, et al. Eur J Endocrinol. 2001 Nov;145(5):645-50. doi: 10.1530/eje.0.1450645. Eur J Endocrinol. 2001. PMID: 11720884

10

Calcium/calmodulin-dependent protein kinase II binds to Raf-1 and modulates integrin-stimulated ERK activation.

Illario M, Cavallo AL, Bayer KU, Di Matola T, Fenzi G, Rossi G, Vitale M. Illario M, et al. Among authors: di matola t. J Biol Chem. 2003 Nov 14;278(46):45101-8. doi: 10.1074/jbc.M305355200. Epub 2003 Sep 3. J Biol Chem. 2003. PMID: 12954639 Free article.

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