Yasukawa T - Search Results

1

Modification defect at anticodon wobble nucleotide of mitochondrial tRNAs(Leu)(UUR) with pathogenic mutations of mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episodes.

Yasukawa T, Suzuki T, Ueda T, Ohta S, Watanabe K. Yasukawa T, et al. J Biol Chem. 2000 Feb 11;275(6):4251-7. doi: 10.1074/jbc.275.6.4251. J Biol Chem. 2000. PMID: 10660592 Free article.

2

Defect in modification at the anticodon wobble nucleotide of mitochondrial tRNA(Lys) with the MERRF encephalomyopathy pathogenic mutation.

Yasukawa T, Suzuki T, Ishii N, Ueda T, Ohta S, Watanabe K. Yasukawa T, et al. FEBS Lett. 2000 Feb 11;467(2-3):175-8. doi: 10.1016/s0014-5793(00)01145-5. FEBS Lett. 2000. PMID: 10675533 Free article.

3

A pathogenic point mutation reduces stability of mitochondrial mutant tRNA(Ile).

Yasukawa T, Hino N, Suzuki T, Watanabe K, Ueda T, Ohta S. Yasukawa T, et al. Nucleic Acids Res. 2000 Oct 1;28(19):3779-84. doi: 10.1093/nar/28.19.3779. Nucleic Acids Res. 2000. PMID: 11000270 Free PMC article.

4

Wobble modification defect in tRNA disturbs codon-anticodon interaction in a mitochondrial disease.

Yasukawa T, Suzuki T, Ishii N, Ohta S, Watanabe K. Yasukawa T, et al. EMBO J. 2001 Sep 3;20(17):4794-802. doi: 10.1093/emboj/20.17.4794. EMBO J. 2001. PMID: 11532943 Free PMC article.

5

The 7472insC mitochondrial DNA mutation impairs the synthesis and extent of aminoacylation of tRNASer(UCN) but not its structure or rate of turnover.

Toompuu M, Yasukawa T, Suzuki T, Hakkinen T, Spelbrink JN, Watanabe K, Jacobs HT. Toompuu M, et al. Among authors: yasukawa t. J Biol Chem. 2002 Jun 21;277(25):22240-50. doi: 10.1074/jbc.M200338200. Epub 2002 Mar 27. J Biol Chem. 2002. PMID: 11919191 Free article.

6

[Wobble modification defect suppresses translational activity of tRNAs with MERRF and MELAS mutations].

Yasukawa T, Suzuki T, Watanabe K, Yasukawa T, Ohta S. Yasukawa T, et al. Nihon Rinsho. 2002 Apr;60 Suppl 4:197-201. Nihon Rinsho. 2002. PMID: 12013848 Review. Japanese. No abstract available.

7

The pathogenic A4269G mutation in human mitochondrial tRNA(Ile) alters the T-stem structure and decreases the binding affinity for elongation factor Tu.

Hino N, Suzuki T, Yasukawa T, Seio K, Watanabe K, Ueda T. Hino N, et al. Among authors: yasukawa t. Genes Cells. 2004 Mar;9(3):243-52. doi: 10.1111/j.1356-9597.2004.00718.x. Genes Cells. 2004. PMID: 15005711 Free article.

8

Codon-specific translational defect caused by a wobble modification deficiency in mutant tRNA from a human mitochondrial disease.

Kirino Y, Yasukawa T, Ohta S, Akira S, Ishihara K, Watanabe K, Suzuki T. Kirino Y, et al. Among authors: yasukawa t. Proc Natl Acad Sci U S A. 2004 Oct 19;101(42):15070-5. doi: 10.1073/pnas.0405173101. Epub 2004 Oct 11. Proc Natl Acad Sci U S A. 2004. PMID: 15477592 Free PMC article.

9

Wobble modification deficiency in mutant tRNAs in patients with mitochondrial diseases.

Yasukawa T, Kirino Y, Ishii N, Holt IJ, Jacobs HT, Makifuchi T, Fukuhara N, Ohta S, Suzuki T, Watanabe K. Yasukawa T, et al. FEBS Lett. 2005 May 23;579(13):2948-52. doi: 10.1016/j.febslet.2005.04.038. FEBS Lett. 2005. PMID: 15893315 Free article.

10

Wobble modification defect suppresses translational activity of tRNAs with MERRF and MELAS mutations.

Yasukawa T, Suzuki T, Ohta S, Watanabe K. Yasukawa T, et al. Mitochondrion. 2002 Nov;2(1-2):129-41. doi: 10.1016/s1567-7249(02)00033-8. Mitochondrion. 2002. PMID: 16120315

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