Maggio JE - Search Results

1

Alzheimer's disease amyloid propagation by a template-dependent dock-lock mechanism.

Esler WP, Stimson ER, Jennings JM, Vinters HV, Ghilardi JR, Lee JP, Mantyh PW, Maggio JE. Esler WP, et al. Among authors: maggio je. Biochemistry. 2000 May 30;39(21):6288-95. doi: 10.1021/bi992933h. Biochemistry. 2000. PMID: 10828941

2

Noninvasive imaging of peripherally injected Alzheimer's disease type synthetic A beta amyloid in vivo.

Marshall JR, Stimson ER, Ghilardi JR, Vinters HV, Mantyh PW, Maggio JE. Marshall JR, et al. Among authors: maggio je. Bioconjug Chem. 2002 Mar-Apr;13(2):276-84. doi: 10.1021/bc010066z. Bioconjug Chem. 2002. PMID: 11906265

3

Apolipoprotein E affects amyloid formation but not amyloid growth in vitro: mechanistic implications for apoE4 enhanced amyloid burden and risk for Alzheimer's disease.

Esler WP, Marshall JR, Stimson ER, Ghilardi JR, Vinters HV, Mantyh PW, Maggio JE. Esler WP, et al. Among authors: maggio je. Amyloid. 2002 Mar;9(1):1-12. doi: 10.3109/13506120209072439. Amyloid. 2002. PMID: 12000192

4

Reversible in vitro growth of Alzheimer disease beta-amyloid plaques by deposition of labeled amyloid peptide.

Maggio JE, Stimson ER, Ghilardi JR, Allen CJ, Dahl CE, Whitcomb DC, Vigna SR, Vinters HV, Labenski ME, Mantyh PW. Maggio JE, et al. Proc Natl Acad Sci U S A. 1992 Jun 15;89(12):5462-6. doi: 10.1073/pnas.89.12.5462. Proc Natl Acad Sci U S A. 1992. PMID: 1608956 Free PMC article.

5

In vitro growth of Alzheimer's disease beta-amyloid plaques displays first-order kinetics.

Esler WP, Stimson ER, Ghilardi JR, Vinters HV, Lee JP, Mantyh PW, Maggio JE. Esler WP, et al. Among authors: maggio je. Biochemistry. 1996 Jan 23;35(3):749-57. doi: 10.1021/bi951685w. Biochemistry. 1996. PMID: 8547255

6

Brain amyloid--a physicochemical perspective.

Maggio JE, Mantyh PW. Maggio JE, et al. Brain Pathol. 1996 Apr;6(2):147-62. doi: 10.1111/j.1750-3639.1996.tb00797.x. Brain Pathol. 1996. PMID: 8737930 Review.

7

Point substitution in the central hydrophobic cluster of a human beta-amyloid congener disrupts peptide folding and abolishes plaque competence.

Esler WP, Stimson ER, Ghilardi JR, Lu YA, Felix AM, Vinters HV, Mantyh PW, Lee JP, Maggio JE. Esler WP, et al. Among authors: maggio je. Biochemistry. 1996 Nov 5;35(44):13914-21. doi: 10.1021/bi961302+. Biochemistry. 1996. PMID: 8909288

8

Deposition of monomeric, not oligomeric, Abeta mediates growth of Alzheimer's disease amyloid plaques in human brain preparations.

Tseng BP, Esler WP, Clish CB, Stimson ER, Ghilardi JR, Vinters HV, Mantyh PW, Lee JP, Maggio JE. Tseng BP, et al. Among authors: maggio je. Biochemistry. 1999 Aug 10;38(32):10424-31. doi: 10.1021/bi990718v. Biochemistry. 1999. PMID: 10441137

9

Activation barriers to structural transition determine deposition rates of Alzheimer's disease a beta amyloid.

Esler WP, Felix AM, Stimson ER, Lachenmann MJ, Ghilardi JR, Lu YA, Vinters HV, Mantyh PW, Lee JP, Maggio JE. Esler WP, et al. Among authors: maggio je. J Struct Biol. 2000 Jun;130(2-3):174-83. doi: 10.1006/jsbi.2000.4276. J Struct Biol. 2000. PMID: 10940224

10

Photoaffinity cross-linking of Alzheimer's disease amyloid fibrils reveals interstrand contact regions between assembled beta-amyloid peptide subunits.

Egnaczyk GF, Greis KD, Stimson ER, Maggio JE. Egnaczyk GF, et al. Among authors: maggio je. Biochemistry. 2001 Oct 2;40(39):11706-14. doi: 10.1021/bi002852h. Biochemistry. 2001. PMID: 11570871

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