Hasegawa A - Search Results

1

72-kDa stress protein (hsp72) induced by administration of dimethylarsinic acid to mice accumulates in alveolar flat cells of lung, a target organ for arsenic carcinogenesis.

Kato K, Yamanaka K, Nakano M, Hasegawa A, Okada S. Kato K, et al. Among authors: hasegawa a. Biol Pharm Bull. 2000 Oct;23(10):1212-5. doi: 10.1248/bpb.23.1212. Biol Pharm Bull. 2000. PMID: 11041253 Free article.

2

DNA single-strand breaks in L-132 cells resulting from inhibition of repair polymerization shortly after exposure to dimethylarsinic acid.

Yamanaka K, Hayashi H, Kato K, Hasegawa A, Oku N, Okada S. Yamanaka K, et al. Among authors: hasegawa a. Biol Pharm Bull. 1997 Feb;20(2):163-7. doi: 10.1248/bpb.20.163. Biol Pharm Bull. 1997. PMID: 9057979 Free article.

3

Cell-nuclear accumulation of 72-kDa stress protein induced by dimethylated arsenics.

Kato K, Yamanaka K, Nakano M, Hasegawa A, Oku N, Okada S. Kato K, et al. Among authors: hasegawa a. Biol Pharm Bull. 1997 Apr;20(4):364-9. doi: 10.1248/bpb.20.364. Biol Pharm Bull. 1997. PMID: 9145211 Free article.

4

Metabolic methylation is a possible genotoxicity-enhancing process of inorganic arsenics.

Yamanaka K, Hayashi H, Tachikawa M, Kato K, Hasegawa A, Oku N, Okada S. Yamanaka K, et al. Among authors: hasegawa a. Mutat Res. 1997 Nov 27;394(1-3):95-101. doi: 10.1016/s1383-5718(97)00130-7. Mutat Res. 1997. PMID: 9434848

5

Dimethylarsinic acid exposure causes accumulation of Hsp72 in cell nuclei and suppresses apoptosis in human alveolar cultured (L-132) cells.

Kato K, Yamanaka K, Hasegawa A, Okada S. Kato K, et al. Among authors: hasegawa a. Biol Pharm Bull. 1999 Nov;22(11):1185-8. doi: 10.1248/bpb.22.1185. Biol Pharm Bull. 1999. PMID: 10598025 Free article.

6

The role of orally administered dimethylarsinic acid, a main metabolite of inorganic arsenics, in the promotion and progression of UVB-induced skin tumorigenesis in hairless mice.

Yamanaka K, Katsumata K, Ikuma K, Hasegawa A, Nakano M, Okada S. Yamanaka K, et al. Among authors: hasegawa a. Cancer Lett. 2000 Apr 28;152(1):79-85. doi: 10.1016/s0304-3835(99)00440-1. Cancer Lett. 2000. PMID: 10754209

7

Oral administration of dimethylarsinic acid, a main metabolite of inorganic arsenic, in mice promotes skin tumorigenesis initiated by dimethylbenz(a)anthracene with or without ultraviolet B as a promoter.

Yamanaka K, Mizol M, Kato K, Hasegawa A, Nakano M, Okada S. Yamanaka K, et al. Among authors: hasegawa a. Biol Pharm Bull. 2001 May;24(5):510-4. doi: 10.1248/bpb.24.510. Biol Pharm Bull. 2001. PMID: 11379771 Free article.

8

Oral exposure of dimethylarsinic acid, a main metabolite of inorganic arsenics, in mice leads to an increase in 8-Oxo-2'-deoxyguanosine level, specifically in the target organs for arsenic carcinogenesis.

Yamanaka K, Takabayashi F, Mizoi M, An Y, Hasegawa A, Okada S. Yamanaka K, et al. Among authors: hasegawa a. Biochem Biophys Res Commun. 2001 Sep 14;287(1):66-70. doi: 10.1006/bbrc.2001.5551. Biochem Biophys Res Commun. 2001. PMID: 11549254

9

Preferential increase of heterochromatin in venular endothelium of lung in mice after administration of dimethylarsinic acid, a major metabolite of inorganic arsenics.

Nakano M, Yamanaka K, Hasegawa A, Sawamura R, Okada S. Nakano M, et al. Among authors: hasegawa a. Carcinogenesis. 1992 Mar;13(3):391-3. doi: 10.1093/carcin/13.3.391. Carcinogenesis. 1992. PMID: 1547528

10

Cellular response to oxidative damage in lung induced by the administration of dimethylarsinic acid, a major metabolite of inorganic arsenics, in mice.

Yamanaka K, Hasegawa A, Sawamura R, Okada S. Yamanaka K, et al. Among authors: hasegawa a. Toxicol Appl Pharmacol. 1991 Apr;108(2):205-13. doi: 10.1016/0041-008x(91)90111-q. Toxicol Appl Pharmacol. 1991. PMID: 2017750

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