Adam-Vizi V - Search Results

1

Mitochondria deficient in complex I activity are depolarized by hydrogen peroxide in nerve terminals: relevance to Parkinson's disease.

Chinopoulos C, Adam-Vizi V. Chinopoulos C, et al. J Neurochem. 2001 Jan;76(1):302-6. doi: 10.1046/j.1471-4159.2001.00060.x. J Neurochem. 2001. PMID: 11146003 Free article.

2

Inhibition of glutamate-induced delayed calcium deregulation by 2-APB and La3+ in cultured cortical neurones.

Chinopoulos C, Gerencser AA, Doczi J, Fiskum G, Adam-Vizi V. Chinopoulos C, et al. J Neurochem. 2004 Oct;91(2):471-83. doi: 10.1111/j.1471-4159.2004.02732.x. J Neurochem. 2004. PMID: 15447680 Free article.

3

Bioenergetics and the formation of mitochondrial reactive oxygen species.

Adam-Vizi V, Chinopoulos C. Adam-Vizi V, et al. Trends Pharmacol Sci. 2006 Dec;27(12):639-45. doi: 10.1016/j.tips.2006.10.005. Epub 2006 Oct 23. Trends Pharmacol Sci. 2006. PMID: 17056127 Review.

4

Enhanced depolarization-evoked calcium signal and reduced [ATP]/[ADP] ratio are unrelated events induced by oxidative stress in synaptosomes.

Tretter L, Chinopoulos C, Adam-Vizi V. Tretter L, et al. J Neurochem. 1997 Dec;69(6):2529-37. doi: 10.1046/j.1471-4159.1997.69062529.x. J Neurochem. 1997. PMID: 9375686 Free article.

5

Plasma membrane depolarization and disturbed Na+ homeostasis induced by the protonophore carbonyl cyanide-p-trifluoromethoxyphenyl-hydrazon in isolated nerve terminals.

Tretter L, Chinopoulos C, Adam-Vizi V. Tretter L, et al. Mol Pharmacol. 1998 Apr;53(4):734-41. doi: 10.1124/mol.53.4.734. Mol Pharmacol. 1998. PMID: 9547365

6

Depolarization of in situ mitochondria due to hydrogen peroxide-induced oxidative stress in nerve terminals: inhibition of alpha-ketoglutarate dehydrogenase.

Chinopoulos C, Tretter L, Adam-Vizi V. Chinopoulos C, et al. J Neurochem. 1999 Jul;73(1):220-8. doi: 10.1046/j.1471-4159.1999.0730220.x. J Neurochem. 1999. PMID: 10386974 Free article.

7

Depolarization of in situ mitochondria by hydrogen peroxide in nerve terminals.

Chinopoulos C, Adam-Vizi V. Chinopoulos C, et al. Ann N Y Acad Sci. 1999;893:269-72. doi: 10.1111/j.1749-6632.1999.tb07834.x. Ann N Y Acad Sci. 1999. PMID: 10672246 No abstract available.

8

Exacerbated responses to oxidative stress by an Na(+) load in isolated nerve terminals: the role of ATP depletion and rise of [Ca(2+)](i).

Chinopoulos C, Tretter L, Rozsa A, Adam-Vizi V. Chinopoulos C, et al. J Neurosci. 2000 Mar 15;20(6):2094-103. doi: 10.1523/JNEUROSCI.20-06-02094.2000. J Neurosci. 2000. PMID: 10704483 Free PMC article.

9

Reversible depolarization of in situ mitochondria by oxidative stress parallels a decrease in NAD(P)H level in nerve terminals.

Chinopoulos C, Tretter L, Adam-Vizi V. Chinopoulos C, et al. Neurochem Int. 2000 May;36(6):483-8. doi: 10.1016/s0197-0186(99)00161-8. Neurochem Int. 2000. PMID: 10762084

10

Glutamate release by an Na+ load and oxidative stress in nerve terminals: relevance to ischemia/reperfusion.

Tretter L, Adam-Vizi V. Tretter L, et al. J Neurochem. 2002 Nov;83(4):855-62. doi: 10.1046/j.1471-4159.2002.01191.x. J Neurochem. 2002. PMID: 12421357 Free article.

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