Su ZZ - Search Results

1

Ionizing radiation modulates vascular endothelial growth factor (VEGF) expression through multiple mitogen activated protein kinase dependent pathways.

Park JS, Qiao L, Su ZZ, Hinman D, Willoughby K, McKinstry R, Yacoub A, Duigou GJ, Young CS, Grant S, Hagan MP, Ellis E, Fisher PB, Dent P. Park JS, et al. Among authors: su zz. Oncogene. 2001 May 31;20(25):3266-80. doi: 10.1038/sj.onc.1204258. Oncogene. 2001. PMID: 11423976

2

Inhibition of AP-1 by SARI negatively regulates transformation progression mediated by CCN1.

Dash R, Su ZZ, Lee SG, Azab B, Boukerche H, Sarkar D, Fisher PB. Dash R, et al. Among authors: su zz. Oncogene. 2010 Aug 5;29(31):4412-23. doi: 10.1038/onc.2010.194. Epub 2010 Jun 7. Oncogene. 2010. PMID: 20531301

3

Ionizing radiation enhances therapeutic activity of mda-7/IL-24: overcoming radiation- and mda-7/IL-24-resistance in prostate cancer cells overexpressing the antiapoptotic proteins bcl-xL or bcl-2.

Su ZZ, Lebedeva IV, Sarkar D, Emdad L, Gupta P, Kitada S, Dent P, Reed JC, Fisher PB. Su ZZ, et al. Oncogene. 2006 Apr 13;25(16):2339-48. doi: 10.1038/sj.onc.1209271. Oncogene. 2006. PMID: 16331261

4

Astrocyte elevated gene-1 activates cell survival pathways through PI3K-Akt signaling.

Lee SG, Su ZZ, Emdad L, Sarkar D, Franke TF, Fisher PB. Lee SG, et al. Among authors: su zz. Oncogene. 2008 Feb 14;27(8):1114-21. doi: 10.1038/sj.onc.1210713. Epub 2007 Aug 20. Oncogene. 2008. PMID: 17704808

5

Astrocyte elevated gene-1 contributes to the pathogenesis of neuroblastoma.

Lee SG, Jeon HY, Su ZZ, Richards JE, Vozhilla N, Sarkar D, Van Maerken T, Fisher PB. Lee SG, et al. Among authors: su zz. Oncogene. 2009 Jul 2;28(26):2476-84. doi: 10.1038/onc.2009.93. Epub 2009 May 18. Oncogene. 2009. PMID: 19448665

6

Src kinase activation is mandatory for MDA-9/syntenin-mediated activation of nuclear factor-kappaB.

Boukerche H, Aissaoui H, Prévost C, Hirbec H, Das SK, Su ZZ, Sarkar D, Fisher PB. Boukerche H, et al. Among authors: su zz. Oncogene. 2010 May 27;29(21):3054-66. doi: 10.1038/onc.2010.65. Epub 2010 Mar 15. Oncogene. 2010. PMID: 20228839 Free PMC article.

7

Cooperation between AP1 and PEA3 sites within the progression elevated gene-3 (PEG-3) promoter regulate basal and differential expression of PEG-3 during progression of the oncogenic phenotype in transformed rat embryo cells.

Su Z, Shi Y, Fisher PB. Su Z, et al. Oncogene. 2000 Jul 13;19(30):3411-21. doi: 10.1038/sj.onc.1203666. Oncogene. 2000. PMID: 10918598

8

PEA3 sites within the progression elevated gene-3 (PEG-3) promoter and mitogen-activated protein kinase contribute to differential PEG-3 expression in Ha-ras and v-raf oncogene transformed rat embryo cells.

Su Z, Shi Y, Friedman R, Qiao L, McKinstry R, Hinman D, Dent P, Fisher PB. Su Z, et al. Nucleic Acids Res. 2001 Apr 15;29(8):1661-71. doi: 10.1093/nar/29.8.1661. Nucleic Acids Res. 2001. PMID: 11292838 Free PMC article.

9

Analysis of viral and cellular gene expression during progression and suppression of the transformed phenotype in type 5 adenovirus-transformed rat embryo cells.

Duigou GJ, Su ZZ, Babiss LE, Driscoll B, Fung YK, Fisher PB. Duigou GJ, et al. Among authors: su zz. Oncogene. 1991 Oct;6(10):1813-24. Oncogene. 1991. PMID: 1923506

10

c-fos is a positive regulator of carcinogen enhancement of adenovirus transformation.

Su ZZ, Yemul S, Stein CA, Fisher PB. Su ZZ, et al. Oncogene. 1995 May 18;10(10):2037-49. Oncogene. 1995. PMID: 7761104

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