Yamamoto M - Search Results

1

Activation of Maf/AP-1 repressor Bach2 by oxidative stress promotes apoptosis and its interaction with promyelocytic leukemia nuclear bodies.

Muto A, Tashiro S, Tsuchiya H, Kume A, Kanno M, Ito E, Yamamoto M, Igarashi K. Muto A, et al. Among authors: yamamoto m. J Biol Chem. 2002 Jun 7;277(23):20724-33. doi: 10.1074/jbc.M112003200. Epub 2002 Mar 28. J Biol Chem. 2002. PMID: 11923289 Free article.

2

Electrophile response element-mediated induction of the cystine/glutamate exchange transporter gene expression.

Sasaki H, Sato H, Kuriyama-Matsumura K, Sato K, Maebara K, Wang H, Tamba M, Itoh K, Yamamoto M, Bannai S. Sasaki H, et al. Among authors: yamamoto m. J Biol Chem. 2002 Nov 22;277(47):44765-71. doi: 10.1074/jbc.M208704200. Epub 2002 Sep 13. J Biol Chem. 2002. PMID: 12235164 Free article.

3

Hemoprotein Bach1 regulates enhancer availability of heme oxygenase-1 gene.

Sun J, Hoshino H, Takaku K, Nakajima O, Muto A, Suzuki H, Tashiro S, Takahashi S, Shibahara S, Alam J, Taketo MM, Yamamoto M, Igarashi K. Sun J, et al. Among authors: yamamoto m. EMBO J. 2002 Oct 1;21(19):5216-24. doi: 10.1093/emboj/cdf516. EMBO J. 2002. PMID: 12356737 Free PMC article.

4

Small Maf compound mutants display central nervous system neuronal degeneration, aberrant transcription, and Bach protein mislocalization coincident with myoclonus and abnormal startle response.

Katsuoka F, Motohashi H, Tamagawa Y, Kure S, Igarashi K, Engel JD, Yamamoto M. Katsuoka F, et al. Among authors: yamamoto m. Mol Cell Biol. 2003 Feb;23(4):1163-74. doi: 10.1128/MCB.23.4.1163-1174.2003. Mol Cell Biol. 2003. PMID: 12556477 Free PMC article.

5

Frequent mutations in the GATA-1 gene in the transient myeloproliferative disorder of Down syndrome.

Xu G, Nagano M, Kanezaki R, Toki T, Hayashi Y, Taketani T, Taki T, Mitui T, Koike K, Kato K, Imaizumi M, Sekine I, Ikeda Y, Hanada R, Sako M, Kudo K, Kojima S, Ohneda O, Yamamoto M, Ito E. Xu G, et al. Among authors: yamamoto m. Blood. 2003 Oct 15;102(8):2960-8. doi: 10.1182/blood-2003-02-0390. Epub 2003 Jun 19. Blood. 2003. PMID: 12816863 Free article.

6

A constitutively active arylhydrocarbon receptor induces growth inhibition of jurkat T cells through changes in the expression of genes related to apoptosis and cell cycle arrest.

Ito T, Tsukumo S, Suzuki N, Motohashi H, Yamamoto M, Fujii-Kuriyama Y, Mimura J, Lin TM, Peterson RE, Tohyama C, Nohara K. Ito T, et al. Among authors: yamamoto m. J Biol Chem. 2004 Jun 11;279(24):25204-10. doi: 10.1074/jbc.M402143200. Epub 2004 Apr 6. J Biol Chem. 2004. PMID: 15069065 Free article.

7

The transcriptional programme of antibody class switching involves the repressor Bach2.

Muto A, Tashiro S, Nakajima O, Hoshino H, Takahashi S, Sakoda E, Ikebe D, Yamamoto M, Igarashi K. Muto A, et al. Among authors: yamamoto m. Nature. 2004 Jun 3;429(6991):566-71. doi: 10.1038/nature02596. Epub 2004 May 19. Nature. 2004. PMID: 15152264 Free article.

8

Oxidative stress sensor Keap1 functions as an adaptor for Cul3-based E3 ligase to regulate proteasomal degradation of Nrf2.

Kobayashi A, Kang MI, Okawa H, Ohtsuji M, Zenke Y, Chiba T, Igarashi K, Yamamoto M. Kobayashi A, et al. Among authors: yamamoto m. Mol Cell Biol. 2004 Aug;24(16):7130-9. doi: 10.1128/MCB.24.16.7130-7139.2004. Mol Cell Biol. 2004. PMID: 15282312 Free PMC article.

9

Nrf2 transcriptionally activates the mafG gene through an antioxidant response element.

Katsuoka F, Motohashi H, Engel JD, Yamamoto M. Katsuoka F, et al. Among authors: yamamoto m. J Biol Chem. 2005 Feb 11;280(6):4483-90. doi: 10.1074/jbc.M411451200. Epub 2004 Dec 1. J Biol Chem. 2005. PMID: 15574414 Free article.

10

Transgenic expression of BACH1 transcription factor results in megakaryocytic impairment.

Toki T, Katsuoka F, Kanezaki R, Xu G, Kurotaki H, Sun J, Kamio T, Watanabe S, Tandai S, Terui K, Yagihashi S, Komatsu N, Igarashi K, Yamamoto M, Ito E. Toki T, et al. Among authors: yamamoto m. Blood. 2005 Apr 15;105(8):3100-8. doi: 10.1182/blood-2004-07-2826. Epub 2004 Dec 21. Blood. 2005. PMID: 15613547 Free article.

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