Kagan BL - Search Results

1

Channel formation by serum amyloid A: a potential mechanism for amyloid pathogenesis and host defense.

Hirakura Y, Carreras I, Sipe JD, Kagan BL. Hirakura Y, et al. Among authors: kagan bl. Amyloid. 2002 Mar;9(1):13-23. doi: 10.3109/13506120209072440. Amyloid. 2002. PMID: 12000193

2

The channel hypothesis of Alzheimer's disease: current status.

Kagan BL, Hirakura Y, Azimov R, Azimova R, Lin MC. Kagan BL, et al. Peptides. 2002 Jul;23(7):1311-5. doi: 10.1016/s0196-9781(02)00067-0. Peptides. 2002. PMID: 12128087 Review.

3

Amyloid peptide channels: blockade by zinc and inhibition by Congo red (amyloid channel block).

Hirakura Y, Yiu WW, Yamamoto A, Kagan BL. Hirakura Y, et al. Among authors: kagan bl. Amyloid. 2000 Sep;7(3):194-9. doi: 10.3109/13506120009146834. Amyloid. 2000. PMID: 11019860

4

Pore formation by beta-2-microglobulin: a mechanism for the pathogenesis of dialysis associated amyloidosis.

Hirakura Y, Kagan BL. Hirakura Y, et al. Among authors: kagan bl. Amyloid. 2001 Jun;8(2):94-100. doi: 10.3109/13506120109007350. Amyloid. 2001. PMID: 11409039

5

Transthyretin channel formation in lipid membranes: implications for pathogenesis.

Hirakura Y, Azimov R, Azimova R, Javier F, Schweitzer ES, Kagan BL. Hirakura Y, et al. Among authors: kagan bl. Amyloid. 2011 Jun;18 Suppl 1:8-10. doi: 10.3109/13506129.2011.574354002. Amyloid. 2011. PMID: 21838414 No abstract available.

6

Alzheimer amyloid abeta1-42 channels: effects of solvent, pH, and Congo Red.

Hirakura Y, Lin MC, Kagan BL. Hirakura Y, et al. Among authors: kagan bl. J Neurosci Res. 1999 Aug 15;57(4):458-66. J Neurosci Res. 1999. PMID: 10440895

7

Polyglutamine-induced ion channels: a possible mechanism for the neurotoxicity of Huntington and other CAG repeat diseases.

Hirakura Y, Azimov R, Azimova R, Kagan BL. Hirakura Y, et al. Among authors: kagan bl. J Neurosci Res. 2000 May 15;60(4):490-4. doi: 10.1002/(sici)1097-4547(20000515)60:4<490::aid-jnr7>3.0.co;2-9. J Neurosci Res. 2000. PMID: 10797551

8

The channel hypothesis of Huntington's disease.

Kagan BL, Hirakura Y, Azimov R, Azimova R. Kagan BL, et al. Brain Res Bull. 2001 Oct-Nov 1;56(3-4):281-4. doi: 10.1016/s0361-9230(01)00654-2. Brain Res Bull. 2001. PMID: 11719262 Review.

9

Membrane perturbation by the neurotoxic Alzheimer amyloid fragment beta 25-35 requires aggregation and beta-sheet formation.

Hirakura Y, Satoh Y, Hirashima N, Suzuki T, Kagan BL, Kirino Y. Hirakura Y, et al. Among authors: kagan bl. Biochem Mol Biol Int. 1998 Nov;46(4):787-94. doi: 10.1080/15216549800204332. Biochem Mol Biol Int. 1998. PMID: 9844740

10

Impairment of hippocampal long-term potentiation by Alzheimer amyloid beta-peptides.

Chen QS, Kagan BL, Hirakura Y, Xie CW. Chen QS, et al. Among authors: kagan bl. J Neurosci Res. 2000 Apr 1;60(1):65-72. doi: 10.1002/(SICI)1097-4547(20000401)60:1<65::AID-JNR7>3.0.CO;2-Q. J Neurosci Res. 2000. PMID: 10723069

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