Maddock HL - Search Results

1

Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning?

Hausenloy DJ, Maddock HL, Baxter GF, Yellon DM. Hausenloy DJ, et al. Among authors: maddock hl. Cardiovasc Res. 2002 Aug 15;55(3):534-43. doi: 10.1016/s0008-6363(02)00455-8. Cardiovasc Res. 2002. PMID: 12160950

2

Adenosine A(3) receptor activation protects the myocardium from reperfusion/reoxygenation injury.

Maddock HL, Mocanu MM, Yellon DM. Maddock HL, et al. Am J Physiol Heart Circ Physiol. 2002 Oct;283(4):H1307-13. doi: 10.1152/ajpheart.00851.2001. Am J Physiol Heart Circ Physiol. 2002. PMID: 12234780 Free article.

3

The cardioprotective and mitochondrial depolarising properties of exogenous nitric oxide in mouse heart.

Bell RM, Maddock HL, Yellon DM. Bell RM, et al. Among authors: maddock hl. Cardiovasc Res. 2003 Feb;57(2):405-15. doi: 10.1016/s0008-6363(02)00675-2. Cardiovasc Res. 2003. PMID: 12566113

4

Myocardial protection from either ischaemic preconditioning or nicorandil is not blocked by gliclazide.

Maddock HL, Siedlecka SM, Yellon DM. Maddock HL, et al. Cardiovasc Drugs Ther. 2004 Mar;18(2):113-9. doi: 10.1023/B:CARD.0000029028.75316.5e. Cardiovasc Drugs Ther. 2004. PMID: 15162072

5

Glimepiride, a novel sulfonylurea, does not abolish myocardial protection afforded by either ischemic preconditioning or diazoxide.

Mocanu MM, Maddock HL, Baxter GF, Lawrence CL, Standen NB, Yellon DM. Mocanu MM, et al. Among authors: maddock hl. Circulation. 2001 Jun 26;103(25):3111-6. doi: 10.1161/01.cir.103.25.3111. Circulation. 2001. PMID: 11425777 Free article.

6

The 10th Biennial Hatter Cardiovascular Institute workshop: cellular protection-evaluating new directions in the setting of myocardial infarction, ischaemic stroke, and cardio-oncology.

Davidson SM, Arjun S, Basalay MV, Bell RM, Bromage DI, Bøtker HE, Carr RD, Cunningham J, Ghosh AK, Heusch G, Ibanez B, Kleinbongard P, Lecour S, Maddock H, Ovize M, Walker M, Wiart M, Yellon DM. Davidson SM, et al. Basic Res Cardiol. 2018 Oct 11;113(6):43. doi: 10.1007/s00395-018-0704-z. Basic Res Cardiol. 2018. PMID: 30310998 Free PMC article.

7

Doxorubicin induced myocardial injury is exacerbated following ischaemic stress via opening of the mitochondrial permeability transition pore.

Gharanei M, Hussain A, Janneh O, Maddock HL. Gharanei M, et al. Among authors: maddock hl. Toxicol Appl Pharmacol. 2013 Apr 15;268(2):149-56. doi: 10.1016/j.taap.2012.12.003. Epub 2012 Dec 20. Toxicol Appl Pharmacol. 2013. PMID: 23262186

8

Caspase inhibition via A3 adenosine receptors: a new cardioprotective mechanism against myocardial infarction.

Hussain A, Gharanei AM, Nagra AS, Maddock HL. Hussain A, et al. Among authors: maddock hl. Cardiovasc Drugs Ther. 2014 Feb;28(1):19-32. doi: 10.1007/s10557-013-6500-y. Cardiovasc Drugs Ther. 2014. PMID: 24220799

9

Role of endothelium in ischaemia-induced myocardial dysfunction of isolated working hearts: cardioprotection by activation of adenosine A(2A) receptors.

Maddock HL, Broadley KJ, Bril A, Khandoudi N. Maddock HL, et al. J Auton Pharmacol. 2001 Oct-Dec;21(5-6):263-71. doi: 10.1046/j.1365-2680.2001.00238.x. J Auton Pharmacol. 2001. PMID: 12123472

10

Attenuation of Sunitinib-induced cardiotoxicity through the A3 adenosine receptor activation.

Sandhu H, Cooper S, Hussain A, Mee C, Maddock H. Sandhu H, et al. Eur J Pharmacol. 2017 Nov 5;814:95-105. doi: 10.1016/j.ejphar.2017.08.011. Epub 2017 Aug 12. Eur J Pharmacol. 2017. PMID: 28811127

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