Gretz N - Search Results

1

Analysis of differential gene expression in stretched podocytes: osteopontin enhances adaptation of podocytes to mechanical stress.

Endlich N, Sunohara M, Nietfeld W, Wolski EW, Schiwek D, Kränzlin B, Gretz N, Kriz W, Eickhoff H, Endlich K. Endlich N, et al. Among authors: gretz n. FASEB J. 2002 Nov;16(13):1850-2. doi: 10.1096/fj.02-0125fje. Epub 2002 Sep 19. FASEB J. 2002. PMID: 12354696

2

Upregulation of Tumor Susceptibility Gene 101 (TSG101) by mechanical stress in podocytes.

Sunohara M, Kriz W, Kränzlin B, Kretzler M, Gretz N, Endlich K, Endlich N. Sunohara M, et al. Among authors: gretz n. Cell Mol Biol (Noisy-le-grand). 2019 Jan 31;65(1):84-88. Cell Mol Biol (Noisy-le-grand). 2019. PMID: 30782301

3

Mechanical stress enhances CD9 expression in cultured podocytes.

Blumenthal A, Giebel J, Warsow G, Li L, Ummanni R, Schordan S, Schordan E, Klemm P, Gretz N, Endlich K, Endlich N. Blumenthal A, et al. Among authors: gretz n. Am J Physiol Renal Physiol. 2015 Mar 15;308(6):F602-13. doi: 10.1152/ajprenal.00190.2014. Epub 2014 Dec 10. Am J Physiol Renal Physiol. 2015. PMID: 25503725 Free article.

4

Pathways to recovery and loss of nephrons in anti-Thy-1 nephritis.

Kriz W, Hähnel B, Hosser H, Ostendorf T, Gaertner S, Kränzlin B, Gretz N, Shimizu F, Floege J. Kriz W, et al. Among authors: gretz n. J Am Soc Nephrol. 2003 Jul;14(7):1904-26. doi: 10.1097/01.asn.0000070073.79690.57. J Am Soc Nephrol. 2003. PMID: 12819253

5

Progression of glomerular diseases: is the podocyte the culprit?

Kriz W, Gretz N, Lemley KV. Kriz W, et al. Among authors: gretz n. Kidney Int. 1998 Sep;54(3):687-97. doi: 10.1046/j.1523-1755.1998.00044.x. Kidney Int. 1998. PMID: 9734594 Free article. Review.

6

Impaired endocytosis may represent an obstacle to gene therapy in polycystic kidney disease.

Witzgall R, Kränzlin B, Gretz N, Obermüller N. Witzgall R, et al. Among authors: gretz n. Kidney Int. 2002 Jan;61(1 Suppl):S132-7. doi: 10.1046/j.1523-1755.2002.0610s1132.x. Kidney Int. 2002. PMID: 11841627 Free article.

7

Urinary clusterin levels in the rat correlate with the severity of tubular damage and may help to differentiate between glomerular and tubular injuries.

Hidaka S, Kränzlin B, Gretz N, Witzgall R. Hidaka S, et al. Among authors: gretz n. Cell Tissue Res. 2002 Dec;310(3):289-96. doi: 10.1007/s00441-002-0629-5. Epub 2002 Oct 2. Cell Tissue Res. 2002. PMID: 12457227

8

Angiotensin II type 1 receptor overexpression in podocytes induces glomerulosclerosis in transgenic rats.

Hoffmann S, Podlich D, Hähnel B, Kriz W, Gretz N. Hoffmann S, et al. Among authors: gretz n. J Am Soc Nephrol. 2004 Jun;15(6):1475-87. doi: 10.1097/01.asn.0000127988.42710.a7. J Am Soc Nephrol. 2004. PMID: 15153558

9

Renal osmotic stress-induced cotransporter: expression in the newborn, adult and post-ischemic rat kidney.

Obermüller N, Kränzlin B, Verma R, Gretz N, Kriz W, Witzgall R. Obermüller N, et al. Among authors: gretz n. Kidney Int. 1997 Dec;52(6):1584-92. doi: 10.1038/ki.1997.489. Kidney Int. 1997. PMID: 9407504 Free article.

10

How does podocyte damage result in tubular damage?

Kriz W, Elger M, Hosser H, Hähnel B, Provoost A, Kränzlin B, Gretz N. Kriz W, et al. Among authors: gretz n. Kidney Blood Press Res. 1999;22(1-2):26-36. doi: 10.1159/000025906. Kidney Blood Press Res. 1999. PMID: 10352405 Review.

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