Li J - Search Results

1

[Mechanisms of extracellular matrix remodeling in dilated cardiomyopathy].

Pauschinger M, Chandrasekharan K, Li J, Schwimmbeck PL, Noutsias M, Schultheiss HP. Pauschinger M, et al. Among authors: li j. Herz. 2002 Nov;27(7):677-82. doi: 10.1007/s00059-002-2413-4. Herz. 2002. PMID: 12439639 Review. German.

2

Collagen degradation in a murine myocarditis model: relevance of matrix metalloproteinase in association with inflammatory induction.

Li J, Schwimmbeck PL, Tschope C, Leschka S, Husmann L, Rutschow S, Reichenbach F, Noutsias M, Kobalz U, Poller W, Spillmann F, Zeichhardt H, Schultheiss HP, Pauschinger M. Li J, et al. Cardiovasc Res. 2002 Nov;56(2):235-47. doi: 10.1016/s0008-6363(02)00546-1. Cardiovasc Res. 2002. PMID: 12393094

3

Carvedilol improves left ventricular function in murine coxsackievirus-induced acute myocarditis association with reduced myocardial interleukin-1beta and MMP-8 expression and a modulated immune response.

Pauschinger M, Rutschow S, Chandrasekharan K, Westermann D, Weitz A, Peter Schwimmbeck L, Zeichhardt H, Poller W, Noutsias M, Li J, Schultheiss HP, Tschope C. Pauschinger M, et al. Among authors: li j. Eur J Heart Fail. 2005 Jun;7(4):444-52. doi: 10.1016/j.ejheart.2004.07.002. Eur J Heart Fail. 2005. PMID: 15921778 Free article.

4

Myocardial proteases and matrix remodeling in inflammatory heart disease.

Rutschow S, Li J, Schultheiss HP, Pauschinger M. Rutschow S, et al. Among authors: li j. Cardiovasc Res. 2006 Feb 15;69(3):646-56. doi: 10.1016/j.cardiores.2005.12.009. Epub 2006 Jan 18. Cardiovasc Res. 2006. PMID: 16417902 Review.

5

Immunomodulation by interleukin-4 suppresses matrix metalloproteinases and improves cardiac function in murine myocarditis.

Li J, Leschka S, Rutschow S, Schwimmbeck PL, Husmann L, Noutsias M, Westermann D, Poller W, Zeichhardt H, Klingel K, Tschope C, Schultheiss HP, Pauschinger M. Li J, et al. Eur J Pharmacol. 2007 Jan 5;554(1):60-8. doi: 10.1016/j.ejphar.2006.08.024. Epub 2006 Aug 26. Eur J Pharmacol. 2007. PMID: 17113076

6

Human apolipoprotein A-I gene transfer reduces the development of experimental diabetic cardiomyopathy.

Van Linthout S, Spillmann F, Riad A, Trimpert C, Lievens J, Meloni M, Escher F, Filenberg E, Demir O, Li J, Shakibaei M, Schimke I, Staudt A, Felix SB, Schultheiss HP, De Geest B, Tschöpe C. Van Linthout S, et al. Among authors: li j. Circulation. 2008 Mar 25;117(12):1563-73. doi: 10.1161/CIRCULATIONAHA.107.710830. Epub 2008 Mar 10. Circulation. 2008. PMID: 18332268

7

The role of the renin-angiotensin-aldosterone system in heart failure.

Unger T, Li J. Unger T, et al. Among authors: li j. J Renin Angiotensin Aldosterone Syst. 2004 Sep;5 Suppl 1:S7-10. doi: 10.3317/jraas.2004.024. J Renin Angiotensin Aldosterone Syst. 2004. PMID: 15526242 Free article. Review.

8

Identification of noncytotoxic and IL-10-producing CD8+AT2R+ T cell population in response to ischemic heart injury.

Curato C, Slavic S, Dong J, Skorska A, Altarche-Xifró W, Miteva K, Kaschina E, Thiel A, Imboden H, Wang J, Steckelings U, Steinhoff G, Unger T, Li J. Curato C, et al. Among authors: li j. J Immunol. 2010 Nov 15;185(10):6286-93. doi: 10.4049/jimmunol.0903681. Epub 2010 Oct 8. J Immunol. 2010. PMID: 20935205

9

Cardiac c-kit+AT2+ cell population is increased in response to ischemic injury and supports cardiomyocyte performance.

Altarche-Xifró W, Curato C, Kaschina E, Grzesiak A, Slavic S, Dong J, Kappert K, Steckelings M, Imboden H, Unger T, Li J. Altarche-Xifró W, et al. Among authors: li j. Stem Cells. 2009 Oct;27(10):2488-97. doi: 10.1002/stem.171. Stem Cells. 2009. PMID: 19591228

10

The CD4(+) AT2R(+) T cell subpopulation improves post-infarction remodelling and restores cardiac function.

Skorska A, von Haehling S, Ludwig M, Lux CA, Gaebel R, Kleiner G, Klopsch C, Dong J, Curato C, Altarche-Xifró W, Slavic S, Unger T, Steinhoff G, Li J, David R. Skorska A, et al. Among authors: li j. J Cell Mol Med. 2015 Aug;19(8):1975-85. doi: 10.1111/jcmm.12574. Epub 2015 May 20. J Cell Mol Med. 2015. PMID: 25991381 Free PMC article.

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