Peschel C - Search Results

1

Inhibition of wild-type and mutant Bcr-Abl by pyrido-pyrimidine-type small molecule kinase inhibitors.

von Bubnoff N, Veach DR, Miller WT, Li W, Sänger J, Peschel C, Bornmann WG, Clarkson B, Duyster J. von Bubnoff N, et al. Among authors: peschel c. Cancer Res. 2003 Oct 1;63(19):6395-404. Cancer Res. 2003. PMID: 14559829

2

BCR-ABL gene mutations in relation to clinical resistance of Philadelphia-chromosome-positive leukaemia to STI571: a prospective study.

von Bubnoff N, Schneller F, Peschel C, Duyster J. von Bubnoff N, et al. Among authors: peschel c. Lancet. 2002 Feb 9;359(9305):487-91. doi: 10.1016/S0140-6736(02)07679-1. Lancet. 2002. PMID: 11853795

3

A cell-based screen for resistance of Bcr-Abl-positive leukemia identifies the mutation pattern for PD166326, an alternative Abl kinase inhibitor.

von Bubnoff N, Veach DR, van der Kuip H, Aulitzky WE, Sänger J, Seipel P, Bornmann WG, Peschel C, Clarkson B, Duyster J. von Bubnoff N, et al. Among authors: peschel c. Blood. 2005 Feb 15;105(4):1652-9. doi: 10.1182/blood-2004-06-2445. Epub 2004 Sep 30. Blood. 2005. PMID: 15459011 Free article.

4

A cell-based screening strategy that predicts mutations in oncogenic tyrosine kinases: implications for clinical resistance in targeted cancer treatment.

von Bubnoff N, Barwisch S, Speicher MR, Peschel C, Duyster J. von Bubnoff N, et al. Among authors: peschel c. Cell Cycle. 2005 Mar;4(3):400-6. doi: 10.4161/cc.4.3.1560. Epub 2005 Mar 25. Cell Cycle. 2005. PMID: 15738656 Review.

5

Bcr-Abl resistance screening predicts a limited spectrum of point mutations to be associated with clinical resistance to the Abl kinase inhibitor nilotinib (AMN107).

von Bubnoff N, Manley PW, Mestan J, Sanger J, Peschel C, Duyster J. von Bubnoff N, et al. Among authors: peschel c. Blood. 2006 Aug 15;108(4):1328-33. doi: 10.1182/blood-2005-12-010132. Epub 2006 Apr 13. Blood. 2006. PMID: 16614241 Free article.

6

The immunogenicity of Bcr-Abl expressing dendritic cells is dependent on the Bcr-Abl kinase activity and dominated by Bcr-Abl regulated antigens.

Scheich F, Duyster J, Peschel C, Bernhard H. Scheich F, et al. Among authors: peschel c. Blood. 2007 Oct 1;110(7):2556-60. doi: 10.1182/blood-2007-01-071001. Epub 2007 Jun 19. Blood. 2007. PMID: 17579186 Free article.

7

Imatinib and leptomycin B are effective in overcoming imatinib-resistance due to Bcr-Abl amplification and clonal evolution but not due to Bcr-Abl kinase domain mutation.

Kancha RK, von Bubnoff N, Miething C, Peschel C, Götze KS, Duyster J. Kancha RK, et al. Among authors: peschel c. Haematologica. 2008 Nov;93(11):1718-22. doi: 10.3324/haematol.13207. Epub 2008 Aug 25. Haematologica. 2008. PMID: 18728023 Free article.

8

Functional analysis of epidermal growth factor receptor (EGFR) mutations and potential implications for EGFR targeted therapy.

Kancha RK, von Bubnoff N, Peschel C, Duyster J. Kancha RK, et al. Among authors: peschel c. Clin Cancer Res. 2009 Jan 15;15(2):460-7. doi: 10.1158/1078-0432.CCR-08-1757. Clin Cancer Res. 2009. PMID: 19147750

9

FMS-like tyrosine kinase 3-internal tandem duplication tyrosine kinase inhibitors display a nonoverlapping profile of resistance mutations in vitro.

von Bubnoff N, Engh RA, Aberg E, Sänger J, Peschel C, Duyster J. von Bubnoff N, et al. Among authors: peschel c. Cancer Res. 2009 Apr 1;69(7):3032-41. doi: 10.1158/0008-5472.CAN-08-2923. Epub 2009 Mar 24. Cancer Res. 2009. PMID: 19318574

10

The epidermal growth factor receptor-L861Q mutation increases kinase activity without leading to enhanced sensitivity toward epidermal growth factor receptor kinase inhibitors.

Kancha RK, Peschel C, Duyster J. Kancha RK, et al. Among authors: peschel c. J Thorac Oncol. 2011 Feb;6(2):387-92. doi: 10.1097/JTO.0b013e3182021f3e. J Thorac Oncol. 2011. PMID: 21252719 Free article.

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