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Tissue production of pro-inflammatory cytokines (IL-1beta, TNFalpha and IL-6) correlates with the intensity of the systemic inflammatory response and with corticosteroid requirements in giant-cell arteritis.
Hernández-Rodríguez J, Segarra M, Vilardell C, Sánchez M, García-Martínez A, Esteban MJ, Queralt C, Grau JM, Urbano-Márquez A, Palacín A, Colomer D, Cid MC. Hernández-Rodríguez J, et al. Among authors: grau jm. Rheumatology (Oxford). 2004 Mar;43(3):294-301. doi: 10.1093/rheumatology/keh058. Epub 2003 Dec 16. Rheumatology (Oxford). 2004. PMID: 14679293
A strong initial systemic inflammatory response is associated with higher corticosteroid requirements and longer duration of therapy in patients with giant-cell arteritis.
Hernández-Rodríguez J, García-Martínez A, Casademont J, Filella X, Esteban MJ, López-Soto A, Fernández-Solà J, Urbano-Márquez A, Grau JM, Cid MC. Hernández-Rodríguez J, et al. Among authors: grau jm. Arthritis Rheum. 2002 Feb;47(1):29-35. doi: 10.1002/art1.10161. Arthritis Rheum. 2002. PMID: 11932875 Free article.
Elevated production of interleukin-6 is associated with a lower incidence of disease-related ischemic events in patients with giant-cell arteritis: angiogenic activity of interleukin-6 as a potential protective mechanism.
Hernández-Rodríguez J, Segarra M, Vilardell C, Sánchez M, García-Martínez A, Esteban MJ, Grau JM, Urbano-Márquez A, Colomer D, Kleinman HK, Cid MC. Hernández-Rodríguez J, et al. Among authors: grau jm. Circulation. 2003 May 20;107(19):2428-34. doi: 10.1161/01.CIR.0000066907.83923.32. Epub 2003 May 12. Circulation. 2003. PMID: 12742994 Clinical Trial.
Gelatinase expression and proteolytic activity in giant-cell arteritis.
Segarra M, García-Martínez A, Sánchez M, Hernández-Rodríguez J, Lozano E, Grau JM, Cid MC. Segarra M, et al. Among authors: grau jm. Ann Rheum Dis. 2007 Nov;66(11):1429-35. doi: 10.1136/ard.2006.068148. Epub 2007 May 14. Ann Rheum Dis. 2007. PMID: 17502363 Free PMC article.
Increased IL-17A expression in temporal artery lesions is a predictor of sustained response to glucocorticoid treatment in patients with giant-cell arteritis.
Espígol-Frigolé G, Corbera-Bellalta M, Planas-Rigol E, Lozano E, Segarra M, García-Martínez A, Prieto-González S, Hernández-Rodríguez J, Grau JM, Rahman MU, Cid MC. Espígol-Frigolé G, et al. Among authors: grau jm. Ann Rheum Dis. 2013 Sep 1;72(9):1481-7. doi: 10.1136/annrheumdis-2012-201836. Epub 2012 Sep 19. Ann Rheum Dis. 2013. PMID: 22993227 Clinical Trial.
Cell adhesion molecules in the development of inflammatory infiltrates in giant cell arteritis: inflammation-induced angiogenesis as the preferential site of leukocyte-endothelial cell interactions.
Cid MC, Cebrián M, Font C, Coll-Vinent B, Hernández-Rodríguez J, Esparza J, Urbano-Márquez A, Grau JM. Cid MC, et al. Among authors: grau jm. Arthritis Rheum. 2000 Jan;43(1):184-94. doi: 10.1002/1529-0131(200001)43:1<184::AID-ANR23>3.0.CO;2-N. Arthritis Rheum. 2000. PMID: 10643715 Free article.
289 results