Kypreos KE - Search Results

1

Probing the pathways of chylomicron and HDL metabolism using adenovirus-mediated gene transfer.

Zannis VI, Chroni A, Kypreos KE, Kan HY, Cesar TB, Zanni EE, Kardassis D. Zannis VI, et al. Among authors: kypreos ke. Curr Opin Lipidol. 2004 Apr;15(2):151-66. doi: 10.1097/00041433-200404000-00008. Curr Opin Lipidol. 2004. PMID: 15017358 Review.

2

Substitutions of glutamate 110 and 111 in the middle helix 4 of human apolipoprotein A-I (apoA-I) by alanine affect the structure and in vitro functions of apoA-I and induce severe hypertriglyceridemia in apoA-I-deficient mice.

Chroni A, Kan HY, Kypreos KE, Gorshkova IN, Shkodrani A, Zannis VI. Chroni A, et al. Among authors: kypreos ke. Biochemistry. 2004 Aug 17;43(32):10442-57. doi: 10.1021/bi049782p. Biochemistry. 2004. PMID: 15301543

3

Pathway of biogenesis of apolipoprotein E-containing HDL in vivo with the participation of ABCA1 and LCAT.

Kypreos KE, Zannis VI. Kypreos KE, et al. Biochem J. 2007 Apr 15;403(2):359-67. doi: 10.1042/BJ20061048. Biochem J. 2007. PMID: 17206937 Free PMC article.

4

Discrete roles of apoA-I and apoE in the biogenesis of HDL species: lessons learned from gene transfer studies in different mouse models.

Zannis VI, Koukos G, Drosatos K, Vezeridis A, Zanni EE, Kypreos KE, Chroni A. Zannis VI, et al. Among authors: kypreos ke. Ann Med. 2008;40 Suppl 1:14-28. doi: 10.1080/07853890701687219. Ann Med. 2008. PMID: 18246469 Free article. Review.

5

Domains of apoE required for binding to apoE receptor 2 and to phospholipids: implications for the functions of apoE in the brain.

Li X, Kypreos K, Zanni EE, Zannis V. Li X, et al. Biochemistry. 2003 Sep 9;42(35):10406-17. doi: 10.1021/bi027093c. Biochemistry. 2003. PMID: 12950167

6

Molecular mechanisms of type III hyperlipoproteinemia: The contribution of the carboxy-terminal domain of ApoE can account for the dyslipidemia that is associated with the E2/E2 phenotype.

Kypreos KE, Li X, van Dijk KW, Havekes LM, Zannis VI. Kypreos KE, et al. Biochemistry. 2003 Aug 26;42(33):9841-53. doi: 10.1021/bi0271796. Biochemistry. 2003. PMID: 12924933

7

Generation of a recombinant apolipoprotein E variant with improved biological functions: hydrophobic residues (LEU-261, TRP-264, PHE-265, LEU-268, VAL-269) of apoE can account for the apoE-induced hypertriglyceridemia.

Kypreos KE, van Dijk KW, Havekes LM, Zannis VI. Kypreos KE, et al. J Biol Chem. 2005 Feb 25;280(8):6276-84. doi: 10.1074/jbc.M413458200. Epub 2004 Dec 2. J Biol Chem. 2005. PMID: 15576362 Free article.

8

SR-BI mediates cholesterol efflux via its interactions with lipid-bound ApoE. Structural mutations in SR-BI diminish cholesterol efflux.

Chroni A, Nieland TJ, Kypreos KE, Krieger M, Zannis VI. Chroni A, et al. Among authors: kypreos ke. Biochemistry. 2005 Oct 4;44(39):13132-43. doi: 10.1021/bi051029o. Biochemistry. 2005. PMID: 16185081

9

LDL receptor deficiency or apoE mutations prevent remnant clearance and induce hypertriglyceridemia in mice.

Kypreos KE, Zannis VI. Kypreos KE, et al. J Lipid Res. 2006 Mar;47(3):521-9. doi: 10.1194/jlr.M500322-JLR200. Epub 2005 Dec 7. J Lipid Res. 2006. PMID: 16339113 Free article.

10

Residues Leu261, Trp264, and Phe265 account for apolipoprotein E-induced dyslipidemia and affect the formation of apolipoprotein E-containing high-density lipoprotein.

Drosatos K, Kypreos KE, Zannis VI. Drosatos K, et al. Among authors: kypreos ke. Biochemistry. 2007 Aug 21;46(33):9645-53. doi: 10.1021/bi700232g. Epub 2007 Jul 27. Biochemistry. 2007. PMID: 17655277 Free PMC article.

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