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Antiphospholipid antibodies from patients with the antiphospholipid syndrome induce monocyte tissue factor expression through the simultaneous activation of NF-kappaB/Rel proteins via the p38 mitogen-activated protein kinase pathway, and of the MEK-1/ERK pathway.
López-Pedrera C, Buendía P, Cuadrado MJ, Siendones E, Aguirre MA, Barbarroja N, Montiel-Duarte C, Torres A, Khamashta M, Velasco F. López-Pedrera C, et al. Among authors: buendia p. Arthritis Rheum. 2006 Jan;54(1):301-11. doi: 10.1002/art.21549. Arthritis Rheum. 2006. PMID: 16385547 Free article.
Inhibition of Flt3-activating mutations does not prevent constitutive activation of ERK/Akt/STAT pathways in some AML cells: a possible cause for the limited effectiveness of monotherapy with small-molecule inhibitors.
Siendones E, Barbarroja N, Torres LA, Buendía P, Velasco F, Dorado G, Torres A, López-Pedrera C. Siendones E, et al. Among authors: buendia p. Hematol Oncol. 2007 Mar;25(1):30-7. doi: 10.1002/hon.805. Hematol Oncol. 2007. PMID: 17128418
Global effects of fluvastatin on the prothrombotic status of patients with antiphospholipid syndrome.
López-Pedrera C, Ruiz-Limón P, Aguirre MÁ, Barbarroja N, Pérez-Sánchez C, Buendía P, Rodriguez-García IC, Rodriguez-Ariza A, Collantes-Estevez E, Velasco F, Khasmahta M, Cuadrado MJ. López-Pedrera C, et al. Among authors: buendia p. Ann Rheum Dis. 2011 Apr;70(4):675-82. doi: 10.1136/ard.2010.135525. Epub 2010 Dec 20. Ann Rheum Dis. 2011. PMID: 21173016
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