Campbell LA - Search Results

1

Lesion progression and plaque composition are not altered in older apoE-/- mice lacking tumor necrosis factor-alpha receptor p55.

Blessing E, Bea F, Kuo CC, Campbell LA, Chesebro B, Rosenfeld ME. Blessing E, et al. Among authors: campbell la. Atherosclerosis. 2004 Oct;176(2):227-32. doi: 10.1016/j.atherosclerosis.2004.05.033. Atherosclerosis. 2004. PMID: 15380444

2

Foam cell formation inhibits growth of Chlamydia pneumoniae but does not attenuate Chlamydia pneumoniae-induced secretion of proinflammatory cytokines.

Blessing E, Kuo CC, Lin TM, Campbell LA, Bea F, Chesebro B, Rosenfeld ME. Blessing E, et al. Among authors: campbell la. Circulation. 2002 Apr 23;105(16):1976-82. doi: 10.1161/01.cir.0000015062.41860.5b. Circulation. 2002. PMID: 11997286

3

Chronic inhibition of cyclooxygenase-2 does not alter plaque composition in a mouse model of advanced unstable atherosclerosis.

Bea F, Blessing E, Bennett BJ, Kuo CC, Campbell LA, Kreuzer J, Rosenfeld ME. Bea F, et al. Among authors: campbell la. Cardiovasc Res. 2003 Oct 15;60(1):198-204. doi: 10.1016/s0008-6363(03)00464-4. Cardiovasc Res. 2003. PMID: 14522423

4

Tumor necrosis factor alpha plays a role in the acceleration of atherosclerosis by Chlamydia pneumoniae in mice.

Campbell LA, Nosaka T, Rosenfeld ME, Yaraei K, Kuo CC. Campbell LA, et al. Infect Immun. 2005 May;73(5):3164-5. doi: 10.1128/IAI.73.5.3164-3165.2005. Infect Immun. 2005. PMID: 15845526 Free PMC article.

5

Chlamydia pneumoniae infection accelerates hyperlipidemia induced atherosclerotic lesion development in C57BL/6J mice.

Blessing E, Campbell LA, Rosenfeld ME, Chough N, Kuo CC. Blessing E, et al. Among authors: campbell la. Atherosclerosis. 2001 Sep;158(1):13-7. doi: 10.1016/s0021-9150(00)00758-9. Atherosclerosis. 2001. PMID: 11500169

6

A 6 week course of azithromycin treatment has no beneficial effect on atherosclerotic lesion development in apolipoprotein E-deficient mice chronically infected with Chlamydia pneumoniae.

Blessing E, Campbell LA, Rosenfeld ME, Chesebro B, Kuo CC. Blessing E, et al. Among authors: campbell la. J Antimicrob Chemother. 2005 Jun;55(6):1037-40. doi: 10.1093/jac/dki128. Epub 2005 Apr 21. J Antimicrob Chemother. 2005. PMID: 15845780

7

Chlamydia pneumoniae induces expression of pro-atherogenic factors through activation of the lectin-like oxidized LDL receptor-1.

Campbell LA, Lee AW, Rosenfeld ME, Kuo CC. Campbell LA, et al. Pathog Dis. 2013 Oct;69(1):1-6. doi: 10.1111/2049-632X.12058. Epub 2013 Oct 1. Pathog Dis. 2013. PMID: 23821487 Free PMC article.

8

Chlamydia pneumoniae and hyperlipidemia are co-risk factors for atherosclerosis: infection prior to induction of hyperlipidemia does not accelerate development of atherosclerotic lesions in C57BL/6J mice.

Blessing E, Campbell LA, Rosenfeld ME, Kuo CC. Blessing E, et al. Among authors: campbell la. Infect Immun. 2002 Sep;70(9):5332-4. doi: 10.1128/IAI.70.9.5332-5334.2002. Infect Immun. 2002. PMID: 12183595 Free PMC article.

9

Chlamydia pneumoniae induces tissue factor expression in mouse macrophages via activation of Egr-1 and the MEK-ERK1/2 pathway.

Bea F, Puolakkainen MH, McMillen T, Hudson FN, Mackman N, Kuo CC, Campbell LA, Rosenfeld ME. Bea F, et al. Among authors: campbell la. Circ Res. 2003 Mar 7;92(4):394-401. doi: 10.1161/01.RES.0000059982.43865.75. Epub 2003 Feb 6. Circ Res. 2003. PMID: 12600889

10

Chlamydia pneumoniae and atherosclerosis.

Campbell LA, Kuo CC. Campbell LA, et al. Semin Respir Infect. 2003 Mar;18(1):48-54. doi: 10.1053/srin.2003.50006. Semin Respir Infect. 2003. PMID: 12652454 Review.

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