Kopp P - Search Results

1

Quantitative trait loci associated with elevated thyroid-stimulating hormone in the Wistar-Kyoto rat.

Baum AE, Solberg LC, Kopp P, Ahmadiyeh N, Churchill G, Takahashi JS, Jameson JL, Redei EE. Baum AE, et al. Among authors: kopp p. Endocrinology. 2005 Feb;146(2):870-8. doi: 10.1210/en.2004-0948. Epub 2004 Oct 28. Endocrinology. 2005. PMID: 15514085 Free PMC article.

2

The thyrotropin (TSH) receptor transmembrane domain mutation (Pro556-Leu) in the hypothyroid hyt/hyt mouse results in plasma membrane targeting but defective TSH binding.

Gu WX, Du GG, Kopp P, Rentoumis A, Albanese C, Kohn LD, Madison LD, Jameson JL. Gu WX, et al. Among authors: kopp p. Endocrinology. 1995 Jul;136(7):3146-53. doi: 10.1210/endo.136.7.7789342. Endocrinology. 1995. PMID: 7789342

3

Thyroid hormone (T3) inhibits ciprofibrate-induced transcription of genes encoding beta-oxidation enzymes: cross talk between peroxisome proliferator and T3 signaling pathways.

Chu R, Madison LD, Lin Y, Kopp P, Rao MS, Jameson JL, Reddy JK. Chu R, et al. Among authors: kopp p. Proc Natl Acad Sci U S A. 1995 Dec 5;92(25):11593-7. doi: 10.1073/pnas.92.25.11593. Proc Natl Acad Sci U S A. 1995. PMID: 8524810 Free PMC article.

4

Syndrome of resistance to thyroid hormone: insights into thyroid hormone action.

Kopp P, Kitajima K, Jameson JL. Kopp P, et al. Proc Soc Exp Biol Med. 1996 Jan;211(1):49-61. doi: 10.3181/00379727-211-43951. Proc Soc Exp Biol Med. 1996. PMID: 8594618 Review.

5

Sexually dimorphic transcriptional responses to gonadotropin-releasing hormone require chronic in vivo exposure to estradiol.

Colin IM, Bauer-Dantoin AC, Sundaresan S, Kopp P, Jameson JL. Colin IM, et al. Among authors: kopp p. Endocrinology. 1996 Jun;137(6):2300-7. doi: 10.1210/endo.137.6.8641179. Endocrinology. 1996. PMID: 8641179

6

Second zinc finger mutants of thyroid hormone receptor selectively preserve DNA binding and heterodimerization but eliminate transcriptional activation.

Nagaya T, Kopp P, Kitajima K, Jameson JL, Seo H. Nagaya T, et al. Among authors: kopp p. Biochem Biophys Res Commun. 1996 May 15;222(2):524-30. doi: 10.1006/bbrc.1996.0777. Biochem Biophys Res Commun. 1996. PMID: 8670238

7

Key elements involved in the negative regulation of the TSH receptor: G protein-coupled receptor kinases, arrestin and inducible cAMP early repressor.

Kopp P. Kopp P. Eur J Endocrinol. 1997 Mar;136(3):269-70. doi: 10.1530/eje.0.1360269. Eur J Endocrinol. 1997. PMID: 9100549 No abstract available.

8

Expression of nitric oxide synthase III in human thyroid follicular cells: evidence for increased expression in hyperthyroidism.

Colin IM, Kopp P, Zbären J, Häberli A, Grizzle WE, Jameson JL. Colin IM, et al. Among authors: kopp p. Eur J Endocrinol. 1997 Jun;136(6):649-55. doi: 10.1530/eje.0.1360649. Eur J Endocrinol. 1997. PMID: 9225730

9

Congenital hyperthyroidism caused by a solitary toxic adenoma harboring a novel somatic mutation (serine281-->isoleucine) in the extracellular domain of the thyrotropin receptor.

Kopp P, Muirhead S, Jourdain N, Gu WX, Jameson JL, Rodd C. Kopp P, et al. J Clin Invest. 1997 Sep 15;100(6):1634-9. doi: 10.1172/JCI119687. J Clin Invest. 1997. PMID: 9294132 Free PMC article.

10

Congenital nonautoimmune hyperthyroidism in a nonidentical twin caused by a sporadic germline mutation in the thyrotropin receptor gene.

Kopp P, Jameson JL, Roe TF. Kopp P, et al. Thyroid. 1997 Oct;7(5):765-70. doi: 10.1089/thy.1997.7.765. Thyroid. 1997. PMID: 9349581

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