Manczak M - Search Results

1

Differential loss of synaptic proteins in Alzheimer's disease: implications for synaptic dysfunction.

Reddy PH, Mani G, Park BS, Jacques J, Murdoch G, Whetsell W Jr, Kaye J, Manczak M. Reddy PH, et al. Among authors: manczak m. J Alzheimers Dis. 2005 Apr;7(2):103-17; discussion 173-80. doi: 10.3233/jad-2005-7203. J Alzheimers Dis. 2005. PMID: 15851848

2

Differential expression of oxidative phosphorylation genes in patients with Alzheimer's disease: implications for early mitochondrial dysfunction and oxidative damage.

Manczak M, Park BS, Jung Y, Reddy PH. Manczak M, et al. Neuromolecular Med. 2004;5(2):147-62. doi: 10.1385/NMM:5:2:147. Neuromolecular Med. 2004. PMID: 15075441

3

Amyloid-beta and mitochondria in aging and Alzheimer's disease: implications for synaptic damage and cognitive decline.

Reddy PH, Manczak M, Mao P, Calkins MJ, Reddy AP, Shirendeb U. Reddy PH, et al. Among authors: manczak m. J Alzheimers Dis. 2010;20 Suppl 2(Suppl 2):S499-512. doi: 10.3233/JAD-2010-100504. J Alzheimers Dis. 2010. PMID: 20413847 Free PMC article. Review.

4

Abnormal interaction between the mitochondrial fission protein Drp1 and hyperphosphorylated tau in Alzheimer's disease neurons: implications for mitochondrial dysfunction and neuronal damage.

Manczak M, Reddy PH. Manczak M, et al. Hum Mol Genet. 2012 Jun 1;21(11):2538-47. doi: 10.1093/hmg/dds072. Epub 2012 Feb 24. Hum Mol Genet. 2012. PMID: 22367970 Free PMC article.

5

Abnormal interaction of VDAC1 with amyloid beta and phosphorylated tau causes mitochondrial dysfunction in Alzheimer's disease.

Manczak M, Reddy PH. Manczak M, et al. Hum Mol Genet. 2012 Dec 1;21(23):5131-46. doi: 10.1093/hmg/dds360. Epub 2012 Aug 27. Hum Mol Genet. 2012. PMID: 22926141 Free PMC article.

6

Abnormal interaction of oligomeric amyloid-β with phosphorylated tau: implications to synaptic dysfunction and neuronal damage.

Manczak M, Reddy PH. Manczak M, et al. J Alzheimers Dis. 2013;36(2):285-95. doi: 10.3233/JAD-130275. J Alzheimers Dis. 2013. PMID: 23594602 Free PMC article.

7

Reduced VDAC1 protects against Alzheimer's disease, mitochondria, and synaptic deficiencies.

Manczak M, Sheiko T, Craigen WJ, Reddy PH. Manczak M, et al. J Alzheimers Dis. 2013;37(4):679-90. doi: 10.3233/JAD-130761. J Alzheimers Dis. 2013. PMID: 23948905 Free PMC article.

8

RNA silencing of genes involved in Alzheimer's disease enhances mitochondrial function and synaptic activity.

Manczak M, Reddy PH. Manczak M, et al. Biochim Biophys Acta. 2013 Dec;1832(12):2368-78. doi: 10.1016/j.bbadis.2013.09.008. Epub 2013 Sep 21. Biochim Biophys Acta. 2013. PMID: 24063855 Free PMC article.

9

Gene expression profiles of transcripts in amyloid precursor protein transgenic mice: up-regulation of mitochondrial metabolism and apoptotic genes is an early cellular change in Alzheimer's disease.

Reddy PH, McWeeney S, Park BS, Manczak M, Gutala RV, Partovi D, Jung Y, Yau V, Searles R, Mori M, Quinn J. Reddy PH, et al. Among authors: manczak m. Hum Mol Genet. 2004 Jun 15;13(12):1225-40. doi: 10.1093/hmg/ddh140. Epub 2004 Apr 28. Hum Mol Genet. 2004. PMID: 15115763

10

Time-course of mitochondrial gene expressions in mice brains: implications for mitochondrial dysfunction, oxidative damage, and cytochrome c in aging.

Manczak M, Jung Y, Park BS, Partovi D, Reddy PH. Manczak M, et al. J Neurochem. 2005 Feb;92(3):494-504. doi: 10.1111/j.1471-4159.2004.02884.x. J Neurochem. 2005. PMID: 15659220 Free article.

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