Park BS - Search Results

1

Differential loss of synaptic proteins in Alzheimer's disease: implications for synaptic dysfunction.

Reddy PH, Mani G, Park BS, Jacques J, Murdoch G, Whetsell W Jr, Kaye J, Manczak M. Reddy PH, et al. Among authors: park bs. J Alzheimers Dis. 2005 Apr;7(2):103-17; discussion 173-80. doi: 10.3233/jad-2005-7203. J Alzheimers Dis. 2005. PMID: 15851848

2

Differential expression of oxidative phosphorylation genes in patients with Alzheimer's disease: implications for early mitochondrial dysfunction and oxidative damage.

Manczak M, Park BS, Jung Y, Reddy PH. Manczak M, et al. Among authors: park bs. Neuromolecular Med. 2004;5(2):147-62. doi: 10.1385/NMM:5:2:147. Neuromolecular Med. 2004. PMID: 15075441

3

Gene expression profiles of transcripts in amyloid precursor protein transgenic mice: up-regulation of mitochondrial metabolism and apoptotic genes is an early cellular change in Alzheimer's disease.

Reddy PH, McWeeney S, Park BS, Manczak M, Gutala RV, Partovi D, Jung Y, Yau V, Searles R, Mori M, Quinn J. Reddy PH, et al. Among authors: park bs. Hum Mol Genet. 2004 Jun 15;13(12):1225-40. doi: 10.1093/hmg/ddh140. Epub 2004 Apr 28. Hum Mol Genet. 2004. PMID: 15115763

4

Time-course of mitochondrial gene expressions in mice brains: implications for mitochondrial dysfunction, oxidative damage, and cytochrome c in aging.

Manczak M, Jung Y, Park BS, Partovi D, Reddy PH. Manczak M, et al. Among authors: park bs. J Neurochem. 2005 Feb;92(3):494-504. doi: 10.1111/j.1471-4159.2004.02884.x. J Neurochem. 2005. PMID: 15659220 Free article.

5

Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression.

Manczak M, Anekonda TS, Henson E, Park BS, Quinn J, Reddy PH. Manczak M, et al. Among authors: park bs. Hum Mol Genet. 2006 May 1;15(9):1437-49. doi: 10.1093/hmg/ddl066. Epub 2006 Mar 21. Hum Mol Genet. 2006. PMID: 16551656

6

Neutralization of granulocyte macrophage colony-stimulating factor decreases amyloid beta 1-42 and suppresses microglial activity in a transgenic mouse model of Alzheimer's disease.

Manczak M, Mao P, Nakamura K, Bebbington C, Park B, Reddy PH. Manczak M, et al. Hum Mol Genet. 2009 Oct 15;18(20):3876-93. doi: 10.1093/hmg/ddp331. Epub 2009 Jul 19. Hum Mol Genet. 2009. PMID: 19617638 Free PMC article.

7

Mitochondria-targeted antioxidants protect against amyloid-beta toxicity in Alzheimer's disease neurons.

Manczak M, Mao P, Calkins MJ, Cornea A, Reddy AP, Murphy MP, Szeto HH, Park B, Reddy PH. Manczak M, et al. J Alzheimers Dis. 2010;20 Suppl 2(Suppl 2):S609-31. doi: 10.3233/JAD-2010-100564. J Alzheimers Dis. 2010. PMID: 20463406 Free PMC article.

8

Toxicity of neurons treated with herbicides and neuroprotection by mitochondria-targeted antioxidant SS31.

Reddy TP, Manczak M, Calkins MJ, Mao P, Reddy AP, Shirendeb U, Park B, Reddy PH. Reddy TP, et al. Int J Environ Res Public Health. 2011 Jan;8(1):203-21. doi: 10.3390/ijerph8010203. Epub 2011 Jan 19. Int J Environ Res Public Health. 2011. PMID: 21318024 Free PMC article.

9

Mitochondrial DNA deletions and differential mitochondrial DNA content in Rhesus monkeys: implications for aging.

Mao P, Gallagher P, Nedungadi S, Manczak M, Shirendeb UP, Kohama SG, Ferguson B, Park BS, Reddy PH. Mao P, et al. Among authors: park bs. Biochim Biophys Acta. 2012 Feb;1822(2):111-9. doi: 10.1016/j.bbadis.2011.10.014. Epub 2011 Oct 28. Biochim Biophys Acta. 2012. PMID: 22056405 Free PMC article.

10

Aging and cytomegalovirus infection differentially and jointly affect distinct circulating T cell subsets in humans.

Wertheimer AM, Bennett MS, Park B, Uhrlaub JL, Martinez C, Pulko V, Currier NL, Nikolich-Žugich D, Kaye J, Nikolich-Žugich J. Wertheimer AM, et al. J Immunol. 2014 Mar 1;192(5):2143-55. doi: 10.4049/jimmunol.1301721. Epub 2014 Feb 5. J Immunol. 2014. PMID: 24501199 Free PMC article. Clinical Trial.

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