Blass JP - Search Results

1

Mitochondrial aconitase is a transglutaminase 2 substrate: transglutamination is a probable mechanism contributing to high-molecular-weight aggregates of aconitase and loss of aconitase activity in Huntington disease brain.

Kim SY, Marekov L, Bubber P, Browne SE, Stavrovskaya I, Lee J, Steinert PM, Blass JP, Beal MF, Gibson GE, Cooper AJ. Kim SY, et al. Among authors: blass jp. Neurochem Res. 2005 Oct;30(10):1245-55. doi: 10.1007/s11064-005-8796-x. Neurochem Res. 2005. PMID: 16341586

2

Transglutaminase activity is present in highly purified nonsynaptosomal mouse brain and liver mitochondria.

Krasnikov BF, Kim SY, McConoughey SJ, Ryu H, Xu H, Stavrovskaya I, Iismaa SE, Mearns BM, Ratan RR, Blass JP, Gibson GE, Cooper AJ. Krasnikov BF, et al. Among authors: blass jp. Biochemistry. 2005 May 31;44(21):7830-43. doi: 10.1021/bi0500877. Biochemistry. 2005. PMID: 15909997 Free PMC article.

3

Mitochondrial enzymes in schizophrenia.

Bubber P, Tang J, Haroutunian V, Xu H, Davis KL, Blass JP, Gibson GE. Bubber P, et al. Among authors: blass jp. J Mol Neurosci. 2004;24(2):315-21. doi: 10.1385/JMN:24:2:315. J Mol Neurosci. 2004. PMID: 15456945

4

Cross linking of polyglutamine domains catalyzed by tissue transglutaminase is greatly favored with pathological-length repeats: does transglutaminase activity play a role in (CAG)(n)/Q(n)-expansion diseases?

Cooper AJ, Jeitner TM, Gentile V, Blass JP. Cooper AJ, et al. Among authors: blass jp. Neurochem Int. 2002 Jan;40(1):53-67. doi: 10.1016/s0197-0186(01)00058-4. Neurochem Int. 2002. PMID: 11738472 Review.

5

Expression in Escherichia coli and purification of hexahistidine-tagged human tissue transglutaminase.

Shi Q, Kim SY, Blass JP, Cooper AJ. Shi Q, et al. Among authors: blass jp. Protein Expr Purif. 2002 Apr;24(3):366-73. doi: 10.1006/prep.2001.1587. Protein Expr Purif. 2002. PMID: 11922752

6

The alpha-ketoglutarate-dehydrogenase complex: a mediator between mitochondria and oxidative stress in neurodegeneration.

Gibson GE, Blass JP, Beal MF, Bunik V. Gibson GE, et al. Among authors: blass jp. Mol Neurobiol. 2005;31(1-3):43-63. doi: 10.1385/MN:31:1-3:043. Mol Neurobiol. 2005. PMID: 15953811 Review.

7

Correlations of disability and biologic alterations in Alzheimer brain and test of significance by a therapeutic trial in humans.

Blass JP, Gibson GE. Blass JP, et al. J Alzheimers Dis. 2006 Jul;9(2):207-18. doi: 10.3233/jad-2006-9212. J Alzheimers Dis. 2006. PMID: 16873967 Clinical Trial.

8

Thiamine-dependent processes and treatment strategies in neurodegeneration.

Gibson GE, Blass JP. Gibson GE, et al. Among authors: blass jp. Antioxid Redox Signal. 2007 Oct;9(10):1605-19. doi: 10.1089/ars.2007.1766. Antioxid Redox Signal. 2007. PMID: 17685850 Review.

9

Cause and consequence: mitochondrial dysfunction initiates and propagates neuronal dysfunction, neuronal death and behavioral abnormalities in age-associated neurodegenerative diseases.

Gibson GE, Starkov A, Blass JP, Ratan RR, Beal MF. Gibson GE, et al. Among authors: blass jp. Biochim Biophys Acta. 2010 Jan;1802(1):122-34. doi: 10.1016/j.bbadis.2009.08.010. Epub 2009 Aug 26. Biochim Biophys Acta. 2010. PMID: 19715758 Free PMC article. Review.

10

The role of the metabolic lesion in Alzheimer's disease.

Blass JP, Gibson GE, Hoyer S. Blass JP, et al. J Alzheimers Dis. 2002 Jun;4(3):225-32. doi: 10.3233/jad-2002-4312. J Alzheimers Dis. 2002. PMID: 12226541 Review.

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