Chapman HD - Search Results

1

Partial versus full allogeneic hemopoietic chimerization is a preferential means to inhibit type 1 diabetes as the latter induces generalized immunosuppression.

Serreze DV, Osborne MA, Chen YG, Chapman HD, Pearson T, Brehm MA, Greiner DL. Serreze DV, et al. Among authors: chapman hd. J Immunol. 2006 Nov 15;177(10):6675-84. doi: 10.4049/jimmunol.177.10.6675. J Immunol. 2006. PMID: 17082580

2

The preferential ability of B lymphocytes to act as diabetogenic APC in NOD mice depends on expression of self-antigen-specific immunoglobulin receptors.

Silveira PA, Johnson E, Chapman HD, Bui T, Tisch RM, Serreze DV. Silveira PA, et al. Among authors: chapman hd. Eur J Immunol. 2002 Dec;32(12):3657-66. doi: 10.1002/1521-4141(200212)32:12<3657::AID-IMMU3657>3.0.CO;2-E. Eur J Immunol. 2002. PMID: 12516557 Free article.

3

Paralytic autoimmune myositis develops in nonobese diabetic mice made Th1 cytokine-deficient by expression of an IFN-gamma receptor beta-chain transgene.

Serreze DV, Pierce MA, Post CM, Chapman HD, Savage H, Bronson RT, Rothman PB, Cox GA. Serreze DV, et al. Among authors: chapman hd. J Immunol. 2003 Mar 1;170(5):2742-9. doi: 10.4049/jimmunol.170.5.2742. J Immunol. 2003. PMID: 12594305

4

B cell selection defects underlie the development of diabetogenic APCs in nonobese diabetic mice.

Silveira PA, Dombrowsky J, Johnson E, Chapman HD, Nemazee D, Serreze DV. Silveira PA, et al. Among authors: chapman hd. J Immunol. 2004 Apr 15;172(8):5086-94. doi: 10.4049/jimmunol.172.8.5086. J Immunol. 2004. PMID: 15067092 Free PMC article.

5

Enhanced pathogenicity of diabetogenic T cells escaping a non-MHC gene-controlled near death experience.

Choisy-Rossi CM, Holl TM, Pierce MA, Chapman HD, Serreze DV. Choisy-Rossi CM, et al. Among authors: chapman hd. J Immunol. 2004 Sep 15;173(6):3791-800. doi: 10.4049/jimmunol.173.6.3791. J Immunol. 2004. PMID: 15356126

6

Activated NKT cells inhibit autoimmune diabetes through tolerogenic recruitment of dendritic cells to pancreatic lymph nodes.

Chen YG, Choisy-Rossi CM, Holl TM, Chapman HD, Besra GS, Porcelli SA, Shaffer DJ, Roopenian D, Wilson SB, Serreze DV. Chen YG, et al. Among authors: chapman hd. J Immunol. 2005 Feb 1;174(3):1196-204. doi: 10.4049/jimmunol.174.3.1196. J Immunol. 2005. PMID: 15661873

7

CD38 is required for the peripheral survival of immunotolerogenic CD4+ invariant NK T cells in nonobese diabetic mice.

Chen YG, Chen J, Osborne MA, Chapman HD, Besra GS, Porcelli SA, Leiter EH, Wilson SB, Serreze DV. Chen YG, et al. Among authors: chapman hd. J Immunol. 2006 Sep 1;177(5):2939-47. doi: 10.4049/jimmunol.177.5.2939. J Immunol. 2006. PMID: 16920929

8

Genes within the Idd5 and Idd9/11 diabetes susceptibility loci affect the pathogenic activity of B cells in nonobese diabetic mice.

Silveira PA, Chapman HD, Stolp J, Johnson E, Cox SL, Hunter K, Wicker LS, Serreze DV. Silveira PA, et al. Among authors: chapman hd. J Immunol. 2006 Nov 15;177(10):7033-41. doi: 10.4049/jimmunol.177.10.7033. J Immunol. 2006. PMID: 17082619 Free PMC article.

9

Cellular expression requirements for inhibition of type 1 diabetes by a dominantly protective major histocompatibility complex haplotype.

Chen YG, Silveira PA, Osborne MA, Chapman HD, Serreze DV. Chen YG, et al. Among authors: chapman hd. Diabetes. 2007 Feb;56(2):424-30. doi: 10.2337/db06-1303. Diabetes. 2007. PMID: 17259387

10

Through regulation of TCR expression levels, an Idd7 region gene(s) interactively contributes to the impaired thymic deletion of autoreactive diabetogenic CD8+ T cells in nonobese diabetic mice.

Serreze DV, Choisy-Rossi CM, Grier AE, Holl TM, Chapman HD, Gahagan JR, Osborne MA, Zhang W, King BL, Brown A, Roopenian D, Marron MP. Serreze DV, et al. Among authors: chapman hd. J Immunol. 2008 Mar 1;180(5):3250-9. doi: 10.4049/jimmunol.180.5.3250. J Immunol. 2008. PMID: 18292549

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