Ma X - Search Results

1

Contribution of recipient-derived cells in allograft neointima formation and the response to stent implantation.

Ma X, Hibbert B, White D, Seymour R, Whitman SC, O'Brien ER. Ma X, et al. PLoS One. 2008 Mar 26;3(3):e1894. doi: 10.1371/journal.pone.0001894. PLoS One. 2008. PMID: 18365026 Free PMC article.

2

Antagonism of the alpha4 integrin subunit attenuates the acute inflammatory response to stent implantation yet is insufficient to prevent late intimal formation.

Ma X, O'Brien ER. Ma X, et al. J Leukoc Biol. 2004 Jun;75(6):1016-21. doi: 10.1189/jlb.1203618. Epub 2004 Mar 12. J Leukoc Biol. 2004. PMID: 15020653

3

Novel antiinflammatory vascular benefits of systemic and stent-based delivery of ethylisopropylamiloride.

Chen YX, Ma X, Whitman S, O'brien ER. Chen YX, et al. Among authors: ma x. Circulation. 2004 Dec 14;110(24):3721-6. doi: 10.1161/01.CIR.0000146790.51331.BA. Epub 2004 Oct 25. Circulation. 2004. PMID: 15505102

4

Inhibition of endothelial progenitor cell glycogen synthase kinase-3beta results in attenuated neointima formation and enhanced re-endothelialization after arterial injury.

Hibbert B, Ma X, Pourdjabbar A, Holm E, Rayner K, Chen YX, Sun J, Filion L, O'Brien ER. Hibbert B, et al. Among authors: ma x. Cardiovasc Res. 2009 Jul 1;83(1):16-23. doi: 10.1093/cvr/cvp156. Epub 2009 May 19. Cardiovasc Res. 2009. PMID: 19454488

5

Delayed re-endothelialization with rapamycin-coated stents is rescued by the addition of a glycogen synthase kinase-3beta inhibitor.

Ma X, Hibbert B, Dhaliwal B, Seibert T, Chen YX, Zhao X, O'Brien ER. Ma X, et al. Cardiovasc Res. 2010 May 1;86(2):338-45. doi: 10.1093/cvr/cvq047. Epub 2010 Feb 17. Cardiovasc Res. 2010. PMID: 20167573

6

Pre-procedural atorvastatin mobilizes endothelial progenitor cells: clues to the salutary effects of statins on healing of stented human arteries.

Hibbert B, Ma X, Pourdjabbar A, Simard T, Rayner K, Sun J, Chen YX, Filion L, O'Brien ER. Hibbert B, et al. Among authors: ma x. PLoS One. 2011 Jan 25;6(1):e16413. doi: 10.1371/journal.pone.0016413. PLoS One. 2011. PMID: 21283543 Free PMC article. Clinical Trial.

7

Pathogenesis of neointima formation following vascular injury.

O' Brien ER, Ma X, Simard T, Pourdjabbar A, Hibbert B. O' Brien ER, et al. Among authors: ma x. Cardiovasc Hematol Disord Drug Targets. 2011 Mar 1;11(1):30-9. doi: 10.2174/187152911795945169. Cardiovasc Hematol Disord Drug Targets. 2011. PMID: 21446903 Review.

8

Heat shock protein 27 attenuates neointima formation and accelerates reendothelialization after arterial injury and stent implantation: importance of vascular endothelial growth factor up-regulation.

Ma X, Hibbert B, McNulty M, Hu T, Zhao X, Ramirez FD, Simard T, de Belleroche JS, O'Brien ER. Ma X, et al. FASEB J. 2014 Feb;28(2):594-602. doi: 10.1096/fj.13-230417. Epub 2013 Oct 18. FASEB J. 2014. PMID: 24142570

9

Current understanding of in-stent restenosis and the potential benefit of drug eluting stents.

Froeschl M, Olsen S, Ma X, O'Brien ER. Froeschl M, et al. Among authors: ma x. Curr Drug Targets Cardiovasc Haematol Disord. 2004 Mar;4(1):103-17. doi: 10.2174/1568006043481293. Curr Drug Targets Cardiovasc Haematol Disord. 2004. PMID: 15032655 Review.

10

Human in-stent restenosis tissue obtained by means of coronary atherectomy consists of an abundant proteoglycan matrix with a paucity of cell proliferation.

Glover C, Ma X, Chen YX, Miller H, Veinot J, Labinaz M, O'Brien E. Glover C, et al. Among authors: ma x. Am Heart J. 2002 Oct;144(4):702-9. doi: 10.1067/mhj.2002.123577. Am Heart J. 2002. PMID: 12360168

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