Ben-Nun A - Search Results

1

Activation and control of pathogenic T cells in OSP/claudin-11-induced EAE in SJL/J mice are dominated by their focused recognition of a single epitopic residue (OSP58M).

Kaushansky N, Eisenstein M, Oved JH, Ben-Nun A. Kaushansky N, et al. Int Immunol. 2008 Nov;20(11):1439-49. doi: 10.1093/intimm/dxn099. Epub 2008 Sep 18. Int Immunol. 2008. PMID: 18801757

2

Anatomy of T cell autoimmunity to myelin oligodendrocyte glycoprotein (MOG): prime role of MOG44F in selection and control of MOG-reactive T cells in H-2b mice.

Ben-Nun A, Kerlero de Rosbo N, Kaushansky N, Eisenstein M, Cohen L, Kaye JF, Mendel I. Ben-Nun A, et al. Eur J Immunol. 2006 Feb;36(2):478-93. doi: 10.1002/eji.200535363. Eur J Immunol. 2006. PMID: 16453383 Free article.

3

Epitope specificity of autoreactive T and B cells associated with experimental autoimmune encephalomyelitis and optic neuritis induced by oligodendrocyte-specific protein in SJL/J mice.

Kaushansky N, Zhong MC, Kerlero de Rosbo N, Hoeftberger R, Lassmann H, Ben-Nun A. Kaushansky N, et al. J Immunol. 2006 Nov 15;177(10):7364-76. doi: 10.4049/jimmunol.177.10.7364. J Immunol. 2006. PMID: 17082656

4

OSP/claudin-11-induced EAE in mice is mediated by pathogenic T cells primarily governed by OSP192Y residue of major encephalitogenic region OSP179-207.

Kaushansky N, Hemo R, Eisenstein M, Ben-Nun A. Kaushansky N, et al. Eur J Immunol. 2007 Jul;37(7):2018-31. doi: 10.1002/eji.200636965. Eur J Immunol. 2007. PMID: 17549734 Free article.

5

Pathogenic T cells in MOBP-induced murine EAE are predominantly focused to recognition of MOBP21F and MOBP27P epitopic residues.

Kaushansky N, Zilkha-Falb R, Hemo R, Lassman H, Eisenstein M, Sas A, Ben-Nun A. Kaushansky N, et al. Eur J Immunol. 2007 Nov;37(11):3281-92. doi: 10.1002/eji.200737438. Eur J Immunol. 2007. PMID: 17935073 Free article.

6

The myelin-associated oligodendrocytic basic protein region MOBP15-36 encompasses the immunodominant major encephalitogenic epitope(s) for SJL/J mice and predicted epitope(s) for multiple sclerosis-associated HLA-DRB1*1501.

de Rosbo NK, Kaye JF, Eisenstein M, Mendel I, Hoeftberger R, Lassmann H, Milo R, Ben-Nun A. de Rosbo NK, et al. J Immunol. 2004 Jul 15;173(2):1426-35. doi: 10.4049/jimmunol.173.2.1426. J Immunol. 2004. PMID: 15240739

7

HLA-DQB1*0602 determines disease susceptibility in a new "humanized" multiple sclerosis model in HLA-DR15 (DRB1*1501;DQB1*0602) transgenic mice.

Kaushansky N, Altmann DM, Ascough S, David CS, Lassmann H, Ben-Nun A. Kaushansky N, et al. J Immunol. 2009 Sep 1;183(5):3531-41. doi: 10.4049/jimmunol.0900784. Epub 2009 Jul 31. J Immunol. 2009. PMID: 19648275

8

The myelin-associated oligodendrocytic basic protein (MOBP) as a relevant primary target autoantigen in multiple sclerosis.

Kaushansky N, Eisenstein M, Zilkha-Falb R, Ben-Nun A. Kaushansky N, et al. Autoimmun Rev. 2010 Feb;9(4):233-6. doi: 10.1016/j.autrev.2009.08.002. Epub 2009 Aug 13. Autoimmun Rev. 2010. PMID: 19683076 Review.

9

'Multi-epitope-targeted' immune-specific therapy for a multiple sclerosis-like disease via engineered multi-epitope protein is superior to peptides.

Kaushansky N, Kerlero de Rosbo N, Zilkha-Falb R, Yosef-Hemo R, Cohen L, Ben-Nun A. Kaushansky N, et al. PLoS One. 2011;6(11):e27860. doi: 10.1371/journal.pone.0027860. Epub 2011 Nov 29. PLoS One. 2011. PMID: 22140475 Free PMC article.

10

DQB1*0602 rather than DRB1*1501 confers susceptibility to multiple sclerosis-like disease induced by proteolipid protein (PLP).

Kaushansky N, Altmann DM, David CS, Lassmann H, Ben-Nun A. Kaushansky N, et al. J Neuroinflammation. 2012 Feb 8;9:29. doi: 10.1186/1742-2094-9-29. J Neuroinflammation. 2012. PMID: 22316121 Free PMC article.

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