Sage E - Search Results

1

The formation of double-strand breaks at multiply damaged sites is driven by the kinetics of excision/incision at base damage in eukaryotic cells.

Kozmin SG, Sedletska Y, Reynaud-Angelin A, Gasparutto D, Sage E. Kozmin SG, et al. Among authors: sage e. Nucleic Acids Res. 2009 Apr;37(6):1767-77. doi: 10.1093/nar/gkp010. Epub 2009 Jan 27. Nucleic Acids Res. 2009. PMID: 19174565 Free PMC article.

2

Roles of Saccharomyces cerevisiae DNA polymerases Poleta and Polzeta in response to irradiation by simulated sunlight.

Kozmin SG, Pavlov YI, Kunkel TA, Sage E. Kozmin SG, et al. Among authors: sage e. Nucleic Acids Res. 2003 Aug 1;31(15):4541-52. doi: 10.1093/nar/gkg489. Nucleic Acids Res. 2003. PMID: 12888515 Free PMC article.

3

Processing of a complex multiply damaged DNA site by human cell extracts and purified repair proteins.

Eot-Houllier G, Eon-Marchais S, Gasparutto D, Sage E. Eot-Houllier G, et al. Among authors: sage e. Nucleic Acids Res. 2005 Jan 12;33(1):260-71. doi: 10.1093/nar/gki165. Print 2005. Nucleic Acids Res. 2005. PMID: 15647508 Free PMC article.

4

Interplay between DNA N-glycosylases/AP lyases at multiply damaged sites and biological consequences.

Eot-Houllier G, Gonera M, Gasparutto D, Giustranti C, Sage E. Eot-Houllier G, et al. Among authors: sage e. Nucleic Acids Res. 2007;35(10):3355-66. doi: 10.1093/nar/gkm190. Epub 2007 Apr 27. Nucleic Acids Res. 2007. PMID: 17468500 Free PMC article.

5

Replication fork collapse is a major cause of the high mutation frequency at three-base lesion clusters.

Sedletska Y, Radicella JP, Sage E. Sedletska Y, et al. Among authors: sage e. Nucleic Acids Res. 2013 Nov;41(20):9339-48. doi: 10.1093/nar/gkt731. Epub 2013 Aug 13. Nucleic Acids Res. 2013. PMID: 23945941 Free PMC article.

6

Dissecting Highly Mutagenic Processing of Complex Clustered DNA Damage in Yeast Saccharomyces cerevisiae.

Kozmin SG, Eot-Houllier G, Reynaud-Angelin A, Gasparutto D, Sage E. Kozmin SG, et al. Among authors: sage e. Cells. 2021 Sep 3;10(9):2309. doi: 10.3390/cells10092309. Cells. 2021. PMID: 34571958 Free PMC article.

7

Bipyrimidine photoproducts rather than oxidative lesions are the main type of DNA damage involved in the genotoxic effect of solar UVA radiation.

Douki T, Reynaud-Angelin A, Cadet J, Sage E. Douki T, et al. Among authors: sage e. Biochemistry. 2003 Aug 5;42(30):9221-6. doi: 10.1021/bi034593c. Biochemistry. 2003. PMID: 12885257

8

UVA radiation is highly mutagenic in cells that are unable to repair 7,8-dihydro-8-oxoguanine in Saccharomyces cerevisiae.

Kozmin S, Slezak G, Reynaud-Angelin A, Elie C, de Rycke Y, Boiteux S, Sage E. Kozmin S, et al. Among authors: sage e. Proc Natl Acad Sci U S A. 2005 Sep 20;102(38):13538-43. doi: 10.1073/pnas.0504497102. Epub 2005 Sep 12. Proc Natl Acad Sci U S A. 2005. PMID: 16157879 Free PMC article.

9

Effect of UVA radiation on membrane fluidity and radical decay in human fibroblasts as detected by spin labeled stearic acids.

Budai M, Reynaud-Angelin A, Szabó Z, Tóth S, Rontó G, Sage E, Gróf P. Budai M, et al. Among authors: sage e. J Photochem Photobiol B. 2004 Dec 2;77(1-3):27-38. doi: 10.1016/j.jphotobiol.2004.08.003. J Photochem Photobiol B. 2004. PMID: 15542359

10

Double-strand break induction and repair in V79-4 hamster cells: the role of core ionisations, as probed by ultrasoft X-rays.

du Penhoat MA, Eschenbrenner A, Abel F, Boissiere A, Guigner JM, Chetioui A, Politis MF, Touati A, Sage E, Jenner TJ, Stevens DL, Hill MA. du Penhoat MA, et al. Among authors: sage e. Int J Radiat Biol. 2010 Mar;86(3):205-19. doi: 10.3109/09553000903419296. Int J Radiat Biol. 2010. PMID: 20201649

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