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Fetal and neonatal exposure to three typical environmental chemicals with different mechanisms of action: mixed exposure to phenol, phthalate, and dioxin cancels the effects of sole exposure on mouse midbrain dopaminergic nuclei.
Tanida T, Warita K, Ishihara K, Fukui S, Mitsuhashi T, Sugawara T, Tabuchi Y, Nanmori T, Qi WM, Inamoto T, Yokoyama T, Kitagawa H, Hoshi N. Tanida T, et al. Toxicol Lett. 2009 Aug 25;189(1):40-7. doi: 10.1016/j.toxlet.2009.04.005. Epub 2009 Apr 11. Toxicol Lett. 2009. PMID: 19481886
When does the sex ratio of offspring of the paternal 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure decrease: in the spermatozoa stage or at fertilization?
Ishihara K, Ohsako S, Tasaka K, Harayama H, Miyake M, Warita K, Tanida T, Mitsuhashi T, Nanmori T, Tabuchi Y, Yokoyama T, Kitagawa H, Hoshi N. Ishihara K, et al. Among authors: tanida t. Reprod Toxicol. 2010 Jan;29(1):68-73. doi: 10.1016/j.reprotox.2009.09.009. Epub 2009 Oct 4. Reprod Toxicol. 2010. PMID: 19808090
Fetal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin transactivates aryl hydrocarbon receptor-responsive element III in the tyrosine hydroxylase immunoreactive neurons of the mouse midbrain.
Tanida T, Tasaka K, Akahoshi E, Ishihara-Sugano M, Saito M, Kawata S, Danjo M, Tokumoto J, Mantani Y, Nagahara D, Tabuchi Y, Yokoyama T, Kitagawa H, Kawata M, Hoshi N. Tanida T, et al. J Appl Toxicol. 2014 Feb;34(2):117-26. doi: 10.1002/jat.2839. Epub 2013 Jan 8. J Appl Toxicol. 2014. PMID: 23296914
160 results