Bayliss DA - Search Results

1

Homeostatic regulation of synaptic excitability: tonic GABA(A) receptor currents replace I(h) in cortical pyramidal neurons of HCN1 knock-out mice.

Chen X, Shu S, Schwartz LC, Sun C, Kapur J, Bayliss DA. Chen X, et al. Among authors: bayliss da. J Neurosci. 2010 Feb 17;30(7):2611-22. doi: 10.1523/JNEUROSCI.3771-09.2010. J Neurosci. 2010. PMID: 20164346 Free PMC article.

2

Postnatal development of serotonergic innervation, 5-HT1A receptor expression, and 5-HT responses in rat motoneurons.

Talley EM, Sadr NN, Bayliss DA. Talley EM, et al. Among authors: bayliss da. J Neurosci. 1997 Jun 1;17(11):4473-85. doi: 10.1523/JNEUROSCI.17-11-04473.1997. J Neurosci. 1997. PMID: 9151764 Free PMC article.

3

Activation of alpha 2-adrenoceptors causes inhibition of calcium channels but does not modulate inwardly-rectifying K+ channels in caudal raphe neurons.

Li YW, Bayliss DA. Li YW, et al. Among authors: bayliss da. Neuroscience. 1998 Feb;82(3):753-65. doi: 10.1016/s0306-4522(97)00312-6. Neuroscience. 1998. PMID: 9483533

4

Electrophysical properties, synaptic transmission and neuromodulation in serotonergic caudal raphe neurons.

Li YW, Bayliss DA. Li YW, et al. Among authors: bayliss da. Clin Exp Pharmacol Physiol. 1998 Jun;25(6):468-73. doi: 10.1111/j.1440-1681.1998.tb02237.x. Clin Exp Pharmacol Physiol. 1998. PMID: 9673827

5

Differential distribution of three members of a gene family encoding low voltage-activated (T-type) calcium channels.

Talley EM, Cribbs LL, Lee JH, Daud A, Perez-Reyes E, Bayliss DA. Talley EM, et al. Among authors: bayliss da. J Neurosci. 1999 Mar 15;19(6):1895-911. doi: 10.1523/JNEUROSCI.19-06-01895.1999. J Neurosci. 1999. PMID: 10066243 Free PMC article.

6

A role for T-type Ca2+ channels in the synergistic control of aldosterone production by ANG II and K+.

Chen XL, Bayliss DA, Fern RJ, Barrett PQ. Chen XL, et al. Among authors: bayliss da. Am J Physiol. 1999 May;276(5):F674-83. doi: 10.1152/ajprenal.1999.276.5.F674. Am J Physiol. 1999. PMID: 10330049

7

The TASK-1 two-pore domain K+ channel is a molecular substrate for neuronal effects of inhalation anesthetics.

Sirois JE, Lei Q, Talley EM, Lynch C 3rd, Bayliss DA. Sirois JE, et al. Among authors: bayliss da. J Neurosci. 2000 Sep 1;20(17):6347-54. doi: 10.1523/JNEUROSCI.20-17-06347.2000. J Neurosci. 2000. PMID: 10964940 Free PMC article.

8

Cns distribution of members of the two-pore-domain (KCNK) potassium channel family.

Talley EM, Solorzano G, Lei Q, Kim D, Bayliss DA. Talley EM, et al. Among authors: bayliss da. J Neurosci. 2001 Oct 1;21(19):7491-505. doi: 10.1523/JNEUROSCI.21-19-07491.2001. J Neurosci. 2001. PMID: 11567039 Free PMC article.

9

Serotonergic raphe neurons express TASK channel transcripts and a TASK-like pH- and halothane-sensitive K+ conductance.

Washburn CP, Sirois JE, Talley EM, Guyenet PG, Bayliss DA. Washburn CP, et al. Among authors: bayliss da. J Neurosci. 2002 Feb 15;22(4):1256-65. doi: 10.1523/JNEUROSCI.22-04-01256.2002. J Neurosci. 2002. PMID: 11850453 Free PMC article.

10

Motoneurons express heteromeric TWIK-related acid-sensitive K+ (TASK) channels containing TASK-1 (KCNK3) and TASK-3 (KCNK9) subunits.

Berg AP, Talley EM, Manger JP, Bayliss DA. Berg AP, et al. Among authors: bayliss da. J Neurosci. 2004 Jul 28;24(30):6693-702. doi: 10.1523/JNEUROSCI.1408-04.2004. J Neurosci. 2004. PMID: 15282272 Free PMC article.

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