Freir DB - Search Results

1

Interaction between prion protein and toxic amyloid β assemblies can be therapeutically targeted at multiple sites.

Freir DB, Nicoll AJ, Klyubin I, Panico S, Mc Donald JM, Risse E, Asante EA, Farrow MA, Sessions RB, Saibil HR, Clarke AR, Rowan MJ, Walsh DM, Collinge J. Freir DB, et al. Nat Commun. 2011 Jun 7;2:336. doi: 10.1038/ncomms1341. Nat Commun. 2011. PMID: 21654636 Free PMC article.

2

Amyloid beta-protein dimers rapidly form stable synaptotoxic protofibrils.

O'Nuallain B, Freir DB, Nicoll AJ, Risse E, Ferguson N, Herron CE, Collinge J, Walsh DM. O'Nuallain B, et al. Among authors: freir db. J Neurosci. 2010 Oct 27;30(43):14411-9. doi: 10.1523/JNEUROSCI.3537-10.2010. J Neurosci. 2010. PMID: 20980598 Free PMC article.

3

Amyloid-β nanotubes are associated with prion protein-dependent synaptotoxicity.

Nicoll AJ, Panico S, Freir DB, Wright D, Terry C, Risse E, Herron CE, O'Malley T, Wadsworth JD, Farrow MA, Walsh DM, Saibil HR, Collinge J. Nicoll AJ, et al. Among authors: freir db. Nat Commun. 2013;4:2416. doi: 10.1038/ncomms3416. Nat Commun. 2013. PMID: 24022506 Free PMC article.

4

Aβ oligomers inhibit synapse remodelling necessary for memory consolidation.

Freir DB, Fedriani R, Scully D, Smith IM, Selkoe DJ, Walsh DM, Regan CM. Freir DB, et al. Neurobiol Aging. 2011 Dec;32(12):2211-8. doi: 10.1016/j.neurobiolaging.2010.01.001. Epub 2010 Jan 25. Neurobiol Aging. 2011. PMID: 20097446 Free PMC article.

5

The Familial British Dementia Mutation Promotes Formation of Neurotoxic Cystine Cross-linked Amyloid Bri (ABri) Oligomers.

Cantlon A, Frigerio CS, Freir DB, Boland B, Jin M, Walsh DM. Cantlon A, et al. Among authors: freir db. J Biol Chem. 2015 Jul 3;290(27):16502-16. doi: 10.1074/jbc.M115.652263. Epub 2015 May 8. J Biol Chem. 2015. PMID: 25957407 Free PMC article.

6

Nicotine enhances the depressive actions of A beta 1-40 on long-term potentiation in the rat hippocampal CA1 region in vivo.

Freir DB, Herron CE. Freir DB, et al. J Neurophysiol. 2003 Jun;89(6):2917-22. doi: 10.1152/jn.00996.2002. Epub 2003 Jan 22. J Neurophysiol. 2003. PMID: 12611941 Free article.

7

Blockade of long-term potentiation by beta-amyloid peptides in the CA1 region of the rat hippocampus in vivo.

Freir DB, Holscher C, Herron CE. Freir DB, et al. J Neurophysiol. 2001 Feb;85(2):708-13. doi: 10.1152/jn.2001.85.2.708. J Neurophysiol. 2001. PMID: 11160505 Free article.

8

Inhibition of LTP in vivo by beta-amyloid peptide in different conformational states.

Schmid AW, Freir DB, Herron CE. Schmid AW, et al. Among authors: freir db. Brain Res. 2008 Mar 4;1197:135-42. doi: 10.1016/j.brainres.2007.11.056. Epub 2007 Dec 4. Brain Res. 2008. PMID: 18237717

9

A beta 25-35-induced depression of long-term potentiation in area CA1 in vivo and in vitro is attenuated by verapamil.

Freir DB, Costello DA, Herron CE. Freir DB, et al. J Neurophysiol. 2003 Jun;89(6):3061-9. doi: 10.1152/jn.00992.2002. Epub 2003 Feb 5. J Neurophysiol. 2003. PMID: 12611943 Free article.

10

Inhibition of L-type voltage dependent calcium channels causes impairment of long-term potentiation in the hippocampal CA1 region in vivo.

Freir DB, Herron CE. Freir DB, et al. Brain Res. 2003 Mar 28;967(1-2):27-36. doi: 10.1016/s0006-8993(02)04190-2. Brain Res. 2003. PMID: 12650963

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