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Carbon monoxide-releasing molecules attenuate postresuscitation myocardial injury and protect cardiac mitochondrial function by reducing the production of mitochondrial reactive oxygen species in a rat model of cardiac arrest.
Yao L, Wang P, Chen M, Liu Y, Zhou L, Fang X, Huang Z. Yao L, et al. Among authors: fang x. J Cardiovasc Pharmacol Ther. 2015 May;20(3):330-41. doi: 10.1177/1074248414559837. Epub 2014 Nov 23. J Cardiovasc Pharmacol Ther. 2015. PMID: 25420477
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