Clark J - Search Results

1

Mechanism and consequence of the autoactivation of p38α mitogen-activated protein kinase promoted by TAB1.

DeNicola GF, Martin ED, Chaikuad A, Bassi R, Clark J, Martino L, Verma S, Sicard P, Tata R, Atkinson RA, Knapp S, Conte MR, Marber MS. DeNicola GF, et al. Among authors: clark j. Nat Struct Mol Biol. 2013 Oct;20(10):1182-90. doi: 10.1038/nsmb.2668. Epub 2013 Sep 15. Nat Struct Mol Biol. 2013. PMID: 24037507 Free PMC article.

2

The TAB1-p38α complex aggravates myocardial injury and can be targeted by small molecules.

De Nicola GF, Bassi R, Nichols C, Fernandez-Caggiano M, Golforoush PA, Thapa D, Anderson R, Martin ED, Verma S, Kleinjung J, Laing A, Hutchinson JP, Eaton P, Clark J, Marber MS. De Nicola GF, et al. Among authors: clark j. JCI Insight. 2018 Aug 23;3(16):e121144. doi: 10.1172/jci.insight.121144. eCollection 2018 Aug 23. JCI Insight. 2018. PMID: 30135318 Free PMC article.

3

The activation of p38 alpha, and not p38 beta, mitogen-activated protein kinase is required for ischemic preconditioning.

Sicard P, Clark JE, Jacquet S, Mohammadi S, Arthur JS, O'Keefe SJ, Marber MS. Sicard P, et al. Among authors: clark je. J Mol Cell Cardiol. 2010 Jun;48(6):1324-8. doi: 10.1016/j.yjmcc.2010.02.013. Epub 2010 Feb 25. J Mol Cell Cardiol. 2010. PMID: 20188737 Free PMC article.

4

MAPKAPK-2 modulates p38-MAPK localization and small heat shock protein phosphorylation but does not mediate the injury associated with p38-MAPK activation during myocardial ischemia.

Gorog DA, Jabr RI, Tanno M, Sarafraz N, Clark JE, Fisher SG, Cao XB, Bellahcene M, Dighe K, Kabir AM, Quinlan RA, Kato K, Gaestel M, Marber MS, Heads RJ. Gorog DA, et al. Among authors: clark je. Cell Stress Chaperones. 2009 Sep;14(5):477-89. doi: 10.1007/s12192-009-0101-5. Epub 2009 Feb 13. Cell Stress Chaperones. 2009. PMID: 19214782 Free PMC article.

5

A chemical genetic approach reveals that p38alpha MAPK activation by diphosphorylation aggravates myocardial infarction and is prevented by the direct binding of SB203580.

Kumphune S, Bassi R, Jacquet S, Sicard P, Clark JE, Verma S, Avkiran M, O'Keefe SJ, Marber MS. Kumphune S, et al. Among authors: clark je. J Biol Chem. 2010 Jan 29;285(5):2968-75. doi: 10.1074/jbc.M109.079228. Epub 2009 Dec 7. J Biol Chem. 2010. PMID: 19996096 Free PMC article.

6

Myocardial stress remodelling after regional infarction is independent of glycogen synthase kinase-3 inactivation.

Webb IG, Sicard P, Clark JE, Redwood S, Marber MS. Webb IG, et al. J Mol Cell Cardiol. 2010 Nov;49(5):897-900. doi: 10.1016/j.yjmcc.2010.07.021. Epub 2010 Aug 6. J Mol Cell Cardiol. 2010. PMID: 20696171 Free PMC article.

7

Advancements in pressure-volume catheter technology - stress remodelling after infarction.

Clark JE, Marber MS. Clark JE, et al. Exp Physiol. 2013 Mar;98(3):614-21. doi: 10.1113/expphysiol.2012.064733. Epub 2012 Oct 12. Exp Physiol. 2013. PMID: 23064506 Free article. Review.

8

Post-infarction remodeling is independent of mitogen-activated protein kinase kinase 3 (MKK3).

Clark JE, Flavell RA, Faircloth ME, Davis RJ, Heads RJ, Marber MS. Clark JE, et al. Cardiovasc Res. 2007 Jun 1;74(3):466-70. doi: 10.1016/j.cardiores.2007.02.027. Epub 2007 Feb 28. Cardiovasc Res. 2007. PMID: 17399693

9

Potential of p38-MAPK inhibitors in the treatment of ischaemic heart disease.

Clark JE, Sarafraz N, Marber MS. Clark JE, et al. Pharmacol Ther. 2007 Nov;116(2):192-206. doi: 10.1016/j.pharmthera.2007.06.013. Epub 2007 Jul 24. Pharmacol Ther. 2007. PMID: 17765316 Review.

10

Targeting p38-MAPK in the ischaemic heart: kill or cure?

Bassi R, Heads R, Marber MS, Clark JE. Bassi R, et al. Curr Opin Pharmacol. 2008 Apr;8(2):141-6. doi: 10.1016/j.coph.2008.01.002. Epub 2008 Mar 4. Curr Opin Pharmacol. 2008. PMID: 18289939 Review.

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