Smith JD - Search Results

1

Site-specific nitration of apolipoprotein A-I at tyrosine 166 is both abundant within human atherosclerotic plaque and dysfunctional.

DiDonato JA, Aulak K, Huang Y, Wagner M, Gerstenecker G, Topbas C, Gogonea V, DiDonato AJ, Tang WHW, Mehl RA, Fox PL, Plow EF, Smith JD, Fisher EA, Hazen SL. DiDonato JA, et al. Among authors: smith jd. J Biol Chem. 2014 Apr 11;289(15):10276-10292. doi: 10.1074/jbc.M114.556506. Epub 2014 Feb 20. J Biol Chem. 2014. PMID: 24558038 Free PMC article.

2

Apolipoprotein A-I is a selective target for myeloperoxidase-catalyzed oxidation and functional impairment in subjects with cardiovascular disease.

Zheng L, Nukuna B, Brennan ML, Sun M, Goormastic M, Settle M, Schmitt D, Fu X, Thomson L, Fox PL, Ischiropoulos H, Smith JD, Kinter M, Hazen SL. Zheng L, et al. Among authors: smith jd. J Clin Invest. 2004 Aug;114(4):529-41. doi: 10.1172/JCI21109. J Clin Invest. 2004. PMID: 15314690 Free PMC article.

3

Localization of nitration and chlorination sites on apolipoprotein A-I catalyzed by myeloperoxidase in human atheroma and associated oxidative impairment in ABCA1-dependent cholesterol efflux from macrophages.

Zheng L, Settle M, Brubaker G, Schmitt D, Hazen SL, Smith JD, Kinter M. Zheng L, et al. Among authors: smith jd. J Biol Chem. 2005 Jan 7;280(1):38-47. doi: 10.1074/jbc.M407019200. Epub 2004 Oct 21. J Biol Chem. 2005. PMID: 15498770 Free article.

4

Tyrosine modification is not required for myeloperoxidase-induced loss of apolipoprotein A-I functional activities.

Peng DQ, Wu Z, Brubaker G, Zheng L, Settle M, Gross E, Kinter M, Hazen SL, Smith JD. Peng DQ, et al. Among authors: smith jd. J Biol Chem. 2005 Oct 7;280(40):33775-84. doi: 10.1074/jbc.M504092200. Epub 2005 Aug 8. J Biol Chem. 2005. PMID: 16091367 Free article.

5

A novel folding intermediate state for apolipoprotein A-I: role of the amino and carboxy termini.

Gross E, Peng DQ, Hazen SL, Smith JD. Gross E, et al. Among authors: smith jd. Biophys J. 2006 Feb 15;90(4):1362-70. doi: 10.1529/biophysj.105.075069. Epub 2005 Dec 2. Biophys J. 2006. PMID: 16326917 Free PMC article.

6

Genetic susceptibility to myocardial infarction and coronary artery disease.

Topol EJ, Smith J, Plow EF, Wang QK. Topol EJ, et al. Hum Mol Genet. 2006 Oct 15;15 Spec No 2:R117-23. doi: 10.1093/hmg/ddl183. Hum Mol Genet. 2006. PMID: 16987874 Review.

7

The refined structure of nascent HDL reveals a key functional domain for particle maturation and dysfunction.

Wu Z, Wagner MA, Zheng L, Parks JS, Shy JM 3rd, Smith JD, Gogonea V, Hazen SL. Wu Z, et al. Among authors: smith jd. Nat Struct Mol Biol. 2007 Sep;14(9):861-8. doi: 10.1038/nsmb1284. Epub 2007 Aug 5. Nat Struct Mol Biol. 2007. PMID: 17676061

8

Association between four SNPs on chromosome 9p21 and myocardial infarction is replicated in an Italian population.

Shen GQ, Rao S, Martinelli N, Li L, Olivieri O, Corrocher R, Abdullah KG, Hazen SL, Smith J, Barnard J, Plow EF, Girelli D, Wang QK. Shen GQ, et al. J Hum Genet. 2008;53(2):144-150. doi: 10.1007/s10038-007-0230-6. Epub 2007 Dec 8. J Hum Genet. 2008. PMID: 18066490

9

Apolipoprotein A-I tryptophan substitution leads to resistance to myeloperoxidase-mediated loss of function.

Peng DQ, Brubaker G, Wu Z, Zheng L, Willard B, Kinter M, Hazen SL, Smith JD. Peng DQ, et al. Among authors: smith jd. Arterioscler Thromb Vasc Biol. 2008 Nov;28(11):2063-70. doi: 10.1161/ATVBAHA.108.173815. Epub 2008 Aug 7. Arterioscler Thromb Vasc Biol. 2008. PMID: 18688016 Free PMC article.

10

Modification of high density lipoprotein by myeloperoxidase generates a pro-inflammatory particle.

Undurti A, Huang Y, Lupica JA, Smith JD, DiDonato JA, Hazen SL. Undurti A, et al. Among authors: smith jd. J Biol Chem. 2009 Nov 6;284(45):30825-35. doi: 10.1074/jbc.M109.047605. Epub 2009 Sep 2. J Biol Chem. 2009. PMID: 19726691 Free PMC article.

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