Mülsch A - Search Results

1

Failure of the sulfhydryl donor N-acetylcysteine (NAC) to reverse nitrate tolerance in large epicardial arteries and the venous capacitance system of the dog.

Münzel T, Holtz J, Mülsch A, Stewart DJ, Bassenge E. Münzel T, et al. Among authors: mulsch a. Z Kardiol. 1989;78 Suppl 2:26-8; discussion 64-7. Z Kardiol. 1989. PMID: 2511688

2

Anti-ischemic actions of molsidomine by venous and large coronary dilatation in combination with antiplatelet effects.

Bassenge E, Mülsch A. Bassenge E, et al. Among authors: mulsch a. J Cardiovasc Pharmacol. 1989;14 Suppl 11:S23-8. J Cardiovasc Pharmacol. 1989. PMID: 2484695

3

Nitrate tolerance in epicardial arteries or in the venous system is not reversed by N-acetylcysteine in vivo, but tolerance-independent interactions exist.

Münzel T, Holtz J, Mülsch A, Stewart DJ, Bassenge E. Münzel T, et al. Among authors: mulsch a. Circulation. 1989 Jan;79(1):188-97. doi: 10.1161/01.cir.79.1.188. Circulation. 1989. PMID: 2562937

4

Clinical tolerance to nitroglycerin is due to impaired biotransformation of nitroglycerin and biological counterregulation, not to desensitization of guanylate cyclase.

Mülsch A, Busse R, Bassenge E. Mülsch A, et al. Z Kardiol. 1989;78 Suppl 2:22-5; discussion 64-7. Z Kardiol. 1989. PMID: 2573982

5

Desensitization of guanylate cyclase in nitrate tolerance does not impair endothelium-dependent responses.

Mülsch A, Busse R, Bassenge E. Mülsch A, et al. Eur J Pharmacol. 1988 Dec 13;158(3):191-8. doi: 10.1016/0014-2999(88)90066-0. Eur J Pharmacol. 1988. PMID: 2908105

6

Mechanisms of interaction between the sulfhydryl precursor L-methionine and glyceryl trinitrate.

Münzel T, Mülsch A, Holtz J, Just H, Harrison DG, Bassenge E. Münzel T, et al. Among authors: mulsch a. Circulation. 1992 Sep;86(3):995-1003. doi: 10.1161/01.cir.86.3.995. Circulation. 1992. PMID: 1355413

7

Effects of native and oxidized low density lipoproteins on formation and inactivation of endothelium-derived relaxing factor.

Galle J, Mülsch A, Busse R, Bassenge E. Galle J, et al. Among authors: mulsch a. Arterioscler Thromb. 1991 Jan-Feb;11(1):198-203. doi: 10.1161/01.atv.11.1.198. Arterioscler Thromb. 1991. PMID: 1987998

8

Modulation of the vasodilator action of SIN-1 by the endothelium.

Busse R, Pohl U, Mülsch A, Bassenge E. Busse R, et al. Among authors: mulsch a. J Cardiovasc Pharmacol. 1989;14 Suppl 11:S81-5. doi: 10.1097/00005344-198906152-00015. J Cardiovasc Pharmacol. 1989. PMID: 2484706

9

LY 83583 (6-anilino-5,8-quinolinedione) blocks nitrovasodilator-induced cyclic GMP increases and inhibition of platelet activation.

Mülsch A, Lückhoff A, Pohl U, Busse R, Bassenge E. Mülsch A, et al. Naunyn Schmiedebergs Arch Pharmacol. 1989 Jul;340(1):119-25. doi: 10.1007/BF00169217. Naunyn Schmiedebergs Arch Pharmacol. 1989. PMID: 2552329

10

Endothelium- and sydnonimine-induced responses of native and cultured aortic smooth muscle cells are not impaired by nitroglycerin tolerance.

Mülsch A, Busse R, Winter I, Bassenge E. Mülsch A, et al. Naunyn Schmiedebergs Arch Pharmacol. 1989 May;339(5):568-74. doi: 10.1007/BF00167263. Naunyn Schmiedebergs Arch Pharmacol. 1989. PMID: 2570362

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