Sugaya M - Search Results

1

An association study of 36 psoriasis susceptibility loci for psoriasis vulgaris and atopic dermatitis in a Japanese population.

Tamari M, Saeki H, Hayashi M, Umezawa Y, Ito T, Fukuchi O, Nobeyama Y, Yanaba K, Nakagawa H, Tsunemi Y, Kato T, Shibata S, Sugaya M, Sato S, Tada Y, Doi S, Miyatake A, Ebe K, Noguchi E, Fujieda S, Ebihara T, Amagai M, Esaki H, Takeuchi S, Furue M, Hirota T. Tamari M, et al. Among authors: sugaya m. J Dermatol Sci. 2014 Nov;76(2):156-7. doi: 10.1016/j.jdermsci.2014.08.005. Epub 2014 Aug 26. J Dermatol Sci. 2014. PMID: 25205357 No abstract available.

2

Both IL-4 and IL-13 inhibit the TNF-alpha and IFN-gamma enhanced MDC production in a human keratinocyte cell line, HaCaT cells.

Xiao T, Kagami S, Saeki H, Sugaya M, Kakinuma T, Fujita H, Yano S, Mitsui H, Torii H, Komine M, Asahina A, Nakamura K, Tamaki K. Xiao T, et al. Among authors: sugaya m. J Dermatol Sci. 2003 Apr;31(2):111-7. doi: 10.1016/s0923-1811(02)00149-4. J Dermatol Sci. 2003. PMID: 12670721

3

Human keratinocytes express fractalkine/CX3CL1.

Sugaya M, Nakamura K, Mitsui H, Takekoshi T, Saeki H, Tamaki K. Sugaya M, et al. J Dermatol Sci. 2003 May;31(3):179-87. doi: 10.1016/s0923-1811(03)00031-8. J Dermatol Sci. 2003. PMID: 12727021

4

Thymus and activation-regulated chemokine (TARC/CCL17) produced by mouse epidermal Langerhans cells is upregulated by TNF-alpha and IL-4 and downregulated by IFN-gamma.

Xiao T, Fujita H, Saeki H, Mitsui H, Sugaya M, Tada Y, Kakinuma T, Torii H, Nakamura K, Asahina A, Tamaki K. Xiao T, et al. Among authors: sugaya m. Cytokine. 2003 Aug 21-Sep 7;23(4-5):126-32. doi: 10.1016/s1043-4666(03)00221-7. Cytokine. 2003. PMID: 12967648

5

Signaling through CD40 ligand decreases CD80 expression on murine Langerhans cells and enhances IL-12 p40 production.

Sugaya M, Nakamura K, Asahina A, Fujita H, Tada Y, Torii H, Tamaki K. Sugaya M, et al. Biochem Biophys Res Commun. 2005 Jun 17;331(4):1045-52. doi: 10.1016/j.bbrc.2005.03.238. Biochem Biophys Res Commun. 2005. PMID: 15882983

6

CCL17 transgenic mice show an enhanced Th2-type response to both allergic and non-allergic stimuli.

Tsunemi Y, Saeki H, Nakamura K, Nagakubo D, Nakayama T, Yoshie O, Kagami S, Shimazu K, Kadono T, Sugaya M, Komine M, Matsushima K, Tamaki K. Tsunemi Y, et al. Among authors: sugaya m. Eur J Immunol. 2006 Aug;36(8):2116-27. doi: 10.1002/eji.200535564. Eur J Immunol. 2006. PMID: 16856206 Free article.

7

IL-17F single nucleotide polymorphism is not associated with psoriasis vulgaris or atopic dermatitis in the Japanese population.

Shibata S, Saeki H, Tsunemi Y, Kato T, Nakamura K, Kakinuma T, Kagami S, Fujita H, Tada Y, Sugaya M, Tamaki K. Shibata S, et al. Among authors: sugaya m. J Dermatol Sci. 2009 Feb;53(2):163-5. doi: 10.1016/j.jdermsci.2008.09.003. Epub 2008 Nov 18. J Dermatol Sci. 2009. PMID: 19019635 No abstract available.

8

Interferon-18 gene polymorphism -137 G/C is associated with susceptibility to psoriasis vulgaris but not with atopic dermatitis in Japanese patients.

Kato T, Tsunemi Y, Saeki H, Shibata S, Sekiya T, Nakamura K, Kakinuma T, Kagami S, Fujita H, Tada Y, Sugaya M, Tamaki K. Kato T, et al. Among authors: sugaya m. J Dermatol Sci. 2009 Feb;53(2):162-3. doi: 10.1016/j.jdermsci.2008.08.016. Epub 2008 Nov 25. J Dermatol Sci. 2009. PMID: 19036565 No abstract available.

9

A case of dermatomyositis with "liver disease associated with rheumatoid diseases" positive for anti-liver-kidney microsome-1 antibody.

Noda S, Asano Y, Tamaki Z, Takekoshi T, Sugaya M, Sato S. Noda S, et al. Among authors: sugaya m. Clin Rheumatol. 2010 Aug;29(8):941-3. doi: 10.1007/s10067-010-1397-3. Epub 2010 Feb 24. Clin Rheumatol. 2010. PMID: 20179980

10

Eotaxins and CCR3 interaction regulates the Th2 environment of cutaneous T-cell lymphoma.

Miyagaki T, Sugaya M, Fujita H, Ohmatsu H, Kakinuma T, Kadono T, Tamaki K, Sato S. Miyagaki T, et al. Among authors: sugaya m. J Invest Dermatol. 2010 Sep;130(9):2304-11. doi: 10.1038/jid.2010.128. Epub 2010 May 27. J Invest Dermatol. 2010. PMID: 20505746 Free article.

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