Chroni A - Search Results

1

Natural human apoA-I mutations L141RPisa and L159RFIN alter HDL structure and functionality and promote atherosclerosis development in mice.

Tiniakou I, Kanaki Z, Georgopoulos S, Chroni A, Van Eck M, Fotakis P, Zannis VI, Kardassis D. Tiniakou I, et al. Among authors: chroni a. Atherosclerosis. 2015 Nov;243(1):77-85. doi: 10.1016/j.atherosclerosis.2015.08.028. Epub 2015 Aug 24. Atherosclerosis. 2015. PMID: 26363436

2

The central helices of ApoA-I can promote ATP-binding cassette transporter A1 (ABCA1)-mediated lipid efflux. Amino acid residues 220-231 of the wild-type ApoA-I are required for lipid efflux in vitro and high density lipoprotein formation in vivo.

Chroni A, Liu T, Gorshkova I, Kan HY, Uehara Y, Von Eckardstein A, Zannis VI. Chroni A, et al. J Biol Chem. 2003 Feb 28;278(9):6719-30. doi: 10.1074/jbc.M205232200. Epub 2002 Dec 17. J Biol Chem. 2003. PMID: 12488454 Free article.

3

ABCA1 and amphipathic apolipoproteins form high-affinity molecular complexes required for cholesterol efflux.

Fitzgerald ML, Morris AL, Chroni A, Mendez AJ, Zannis VI, Freeman MW. Fitzgerald ML, et al. Among authors: chroni a. J Lipid Res. 2004 Feb;45(2):287-94. doi: 10.1194/jlr.M300355-JLR200. Epub 2003 Nov 16. J Lipid Res. 2004. PMID: 14617740 Free article.

4

Cross-linking and lipid efflux properties of apoA-I mutants suggest direct association between apoA-I helices and ABCA1.

Chroni A, Liu T, Fitzgerald ML, Freeman MW, Zannis VI. Chroni A, et al. Biochemistry. 2004 Feb 24;43(7):2126-39. doi: 10.1021/bi035813p. Biochemistry. 2004. PMID: 14967052

5

Probing the pathways of chylomicron and HDL metabolism using adenovirus-mediated gene transfer.

Zannis VI, Chroni A, Kypreos KE, Kan HY, Cesar TB, Zanni EE, Kardassis D. Zannis VI, et al. Among authors: chroni a. Curr Opin Lipidol. 2004 Apr;15(2):151-66. doi: 10.1097/00041433-200404000-00008. Curr Opin Lipidol. 2004. PMID: 15017358 Review.

6

Cross-inhibition of SR-BI- and ABCA1-mediated cholesterol transport by the small molecules BLT-4 and glyburide.

Nieland TJ, Chroni A, Fitzgerald ML, Maliga Z, Zannis VI, Kirchhausen T, Krieger M. Nieland TJ, et al. Among authors: chroni a. J Lipid Res. 2004 Jul;45(7):1256-65. doi: 10.1194/jlr.M300358-JLR200. Epub 2004 Apr 21. J Lipid Res. 2004. PMID: 15102890 Free article.

7

Substitutions of glutamate 110 and 111 in the middle helix 4 of human apolipoprotein A-I (apoA-I) by alanine affect the structure and in vitro functions of apoA-I and induce severe hypertriglyceridemia in apoA-I-deficient mice.

Chroni A, Kan HY, Kypreos KE, Gorshkova IN, Shkodrani A, Zannis VI. Chroni A, et al. Biochemistry. 2004 Aug 17;43(32):10442-57. doi: 10.1021/bi049782p. Biochemistry. 2004. PMID: 15301543

8

Deletions of helices 2 and 3 of human apoA-I are associated with severe dyslipidemia following adenovirus-mediated gene transfer in apoA-I-deficient mice.

Chroni A, Kan HY, Shkodrani A, Liu T, Zannis VI. Chroni A, et al. Biochemistry. 2005 Mar 15;44(10):4108-17. doi: 10.1021/bi047998l. Biochemistry. 2005. PMID: 15751988

9

SR-BI mediates cholesterol efflux via its interactions with lipid-bound ApoE. Structural mutations in SR-BI diminish cholesterol efflux.

Chroni A, Nieland TJ, Kypreos KE, Krieger M, Zannis VI. Chroni A, et al. Biochemistry. 2005 Oct 4;44(39):13132-43. doi: 10.1021/bi051029o. Biochemistry. 2005. PMID: 16185081

10

Point mutations in apolipoprotein A-I mimic the phenotype observed in patients with classical lecithin:cholesterol acyltransferase deficiency.

Chroni A, Duka A, Kan HY, Liu T, Zannis VI. Chroni A, et al. Biochemistry. 2005 Nov 1;44(43):14353-66. doi: 10.1021/bi050962o. Biochemistry. 2005. PMID: 16245952

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