Kurokawa M - Search Results

1

Genetic basis of myeloid transformation in familial platelet disorder/acute myeloid leukemia patients with haploinsufficient RUNX1 allele.

Sakurai M, Kasahara H, Yoshida K, Yoshimi A, Kunimoto H, Watanabe N, Shiraishi Y, Chiba K, Tanaka H, Harada Y, Harada H, Kawakita T, Kurokawa M, Miyano S, Takahashi S, Ogawa S, Okamoto S, Nakajima H. Sakurai M, et al. Among authors: kurokawa m. Blood Cancer J. 2016 Feb 5;6(2):e392. doi: 10.1038/bcj.2015.81. Blood Cancer J. 2016. PMID: 26849013 Free PMC article. No abstract available.

2

The t(3;21) fusion product, AML1/Evi-1, interacts with Smad3 and blocks transforming growth factor-beta-mediated growth inhibition of myeloid cells.

Kurokawa M, Mitani K, Imai Y, Ogawa S, Yazaki Y, Hirai H. Kurokawa M, et al. Blood. 1998 Dec 1;92(11):4003-12. Blood. 1998. PMID: 9834202 Free article.

3

Spectrum of clinical and genetic features of patients with inherited platelet disorder with suspected predisposition to hematological malignancies: a nationwide survey in Japan.

Yoshimi A, Toya T, Nannya Y, Takaoka K, Kirito K, Ito E, Nakajima H, Hayashi Y, Takahashi T, Moriya-Saito A, Suzuki K, Harada H, Komatsu N, Usuki K, Ichikawa M, Kurokawa M. Yoshimi A, et al. Among authors: kurokawa m. Ann Oncol. 2016 May;27(5):887-95. doi: 10.1093/annonc/mdw066. Epub 2016 Feb 15. Ann Oncol. 2016. PMID: 26884589 Free article.

4

The AML1/ETO(MTG8) and AML1/Evi-1 leukemia-associated chimeric oncoproteins accumulate PEBP2beta(CBFbeta) in the nucleus more efficiently than wild-type AML1.

Tanaka K, Tanaka T, Kurokawa M, Imai Y, Ogawa S, Mitani K, Yazaki Y, Hirai H. Tanaka K, et al. Among authors: kurokawa m. Blood. 1998 Mar 1;91(5):1688-99. Blood. 1998. PMID: 9473235 Free article.

5

Mutations of the AML1 gene in myelodysplastic syndrome and their functional implications in leukemogenesis.

Imai Y, Kurokawa M, Izutsu K, Hangaishi A, Takeuchi K, Maki K, Ogawa S, Chiba S, Mitani K, Hirai H. Imai Y, et al. Among authors: kurokawa m. Blood. 2000 Nov 1;96(9):3154-60. Blood. 2000. PMID: 11049997 Free article.

6

Homozygous loss of the cyclin-dependent kinase 4-inhibitor (p16) gene in human leukemias.

Ogawa S, Hirano N, Sato N, Takahashi T, Hangaishi A, Tanaka K, Kurokawa M, Tanaka T, Mitani K, Yazaki Y, et al. Ogawa S, et al. Among authors: kurokawa m. Blood. 1994 Oct 15;84(8):2431-5. Blood. 1994. PMID: 7919362 Free article.

7

Overexpression of the AML1 proto-oncoprotein in NIH3T3 cells leads to neoplastic transformation depending on the DNA-binding and transactivational potencies.

Kurokawa M, Tanaka T, Tanaka K, Ogawa S, Mitani K, Yazaki Y, Hirai H. Kurokawa M, et al. Oncogene. 1996 Feb 15;12(4):883-92. Oncogene. 1996. PMID: 8632911

8

An acute myeloid leukemia gene, AML1, regulates transcriptional activation and hemopoietic myeloid cell differentiation antagonistically by two alternative spliced forms.

Tanaka T, Tanaka K, Ogawa S, Kurokawa M, Mitani K, Yazaki Y, Shibata Y, Hirai H. Tanaka T, et al. Among authors: kurokawa m. Leukemia. 1997 Apr;11 Suppl 3:299-302. Leukemia. 1997. PMID: 9209372

9

An acute myeloid leukemia gene, AML1, regulates hemopoietic myeloid cell differentiation and transcriptional activation antagonistically by two alternative spliced forms.

Tanaka T, Tanaka K, Ogawa S, Kurokawa M, Mitani K, Nishida J, Shibata Y, Yazaki Y, Hirai H. Tanaka T, et al. Among authors: kurokawa m. EMBO J. 1995 Jan 16;14(2):341-50. doi: 10.1002/j.1460-2075.1995.tb07008.x. EMBO J. 1995. PMID: 7530657 Free PMC article.

10

AML1-Evi-1 specifically transforms hematopoietic stem cells through fusion of the entire Evi-1 sequence to AML1.

Takeshita M, Ichikawa M, Nitta E, Goyama S, Asai T, Ogawa S, Chiba S, Kurokawa M. Takeshita M, et al. Among authors: kurokawa m. Leukemia. 2008 Jun;22(6):1241-9. doi: 10.1038/leu.2008.53. Epub 2008 Mar 13. Leukemia. 2008. PMID: 18337762

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