Takahashi C - Search Results

1

The RB-IL-6 axis controls self-renewal and endocrine therapy resistance by fine-tuning mitochondrial activity.

Kitajima S, Yoshida A, Kohno S, Li F, Suzuki S, Nagatani N, Nishimoto Y, Sasaki N, Muranaka H, Wan Y, Thai TC, Okahashi N, Matsuda F, Shimizu H, Nishiuchi T, Suzuki Y, Tominaga K, Gotoh N, Suzuki M, Ewen ME, Barbie DA, Hirose O, Tanaka T, Takahashi C. Kitajima S, et al. Among authors: takahashi c. Oncogene. 2017 Sep 7;36(36):5145-5157. doi: 10.1038/onc.2017.124. Epub 2017 May 8. Oncogene. 2017. PMID: 28481867

2

Characterization of a human MSX-2 cDNA and its fragment isolated as a transformation suppressor gene against v-Ki-ras oncogene.

Takahashi C, Akiyama N, Matsuzaki T, Takai S, Kitayama H, Noda M. Takahashi C, et al. Oncogene. 1996 May 16;12(10):2137-46. Oncogene. 1996. PMID: 8668339

3

Rb and N-ras function together to control differentiation in the mouse.

Takahashi C, Bronson RT, Socolovsky M, Contreras B, Lee KY, Jacks T, Noda M, Kucherlapati R, Ewen ME. Takahashi C, et al. Mol Cell Biol. 2003 Aug;23(15):5256-68. doi: 10.1128/MCB.23.15.5256-5268.2003. Mol Cell Biol. 2003. PMID: 12861012 Free PMC article.

4

Genetic interaction between Rb and K-ras in the control of differentiation and tumor suppression.

Takahashi C, Contreras B, Bronson RT, Loda M, Ewen ME. Takahashi C, et al. Mol Cell Biol. 2004 Dec;24(23):10406-15. doi: 10.1128/MCB.24.23.10406-10415.2004. Mol Cell Biol. 2004. PMID: 15542848 Free PMC article.

5

Nras loss induces metastatic conversion of Rb1-deficient neuroendocrine thyroid tumor.

Takahashi C, Contreras B, Iwanaga T, Takegami Y, Bakker A, Bronson RT, Noda M, Loda M, Hunt JL, Ewen ME. Takahashi C, et al. Nat Genet. 2006 Jan;38(1):118-23. doi: 10.1038/ng1703. Epub 2005 Dec 20. Nat Genet. 2006. PMID: 16369533

6

Genetic interaction between Rb and N-ras: differentiation control and metastasis.

Takahashi C, Ewen ME. Takahashi C, et al. Cancer Res. 2006 Oct 1;66(19):9345-8. doi: 10.1158/0008-5472.CAN-06-1250. Cancer Res. 2006. PMID: 17018584 Review.

7

Wild-type NRas and KRas perform distinct functions during transformation.

Fotiadou PP, Takahashi C, Rajabi HN, Ewen ME. Fotiadou PP, et al. Among authors: takahashi c. Mol Cell Biol. 2007 Oct;27(19):6742-55. doi: 10.1128/MCB.00234-07. Epub 2007 Jul 16. Mol Cell Biol. 2007. PMID: 17636015 Free PMC article.

8

Rb Regulates DNA damage response and cellular senescence through E2F-dependent suppression of N-ras isoprenylation.

Shamma A, Takegami Y, Miki T, Kitajima S, Noda M, Obara T, Okamoto T, Takahashi C. Shamma A, et al. Among authors: takahashi c. Cancer Cell. 2009 Apr 7;15(4):255-69. doi: 10.1016/j.ccr.2009.03.001. Cancer Cell. 2009. PMID: 19345325 Free article.

9

Hypoxia and RAS-signaling pathways converge on, and cooperatively downregulate, the RECK tumor-suppressor protein through microRNAs.

Loayza-Puch F, Yoshida Y, Matsuzaki T, Takahashi C, Kitayama H, Noda M. Loayza-Puch F, et al. Among authors: takahashi c. Oncogene. 2010 May 6;29(18):2638-48. doi: 10.1038/onc.2010.23. Epub 2010 Feb 15. Oncogene. 2010. PMID: 20154725

10

The ß1-integrin-dependent function of RECK in physiologic and tumor angiogenesis.

Miki T, Shamma A, Kitajima S, Takegami Y, Noda M, Nakashima Y, Watanabe K, Takahashi C. Miki T, et al. Among authors: takahashi c. Mol Cancer Res. 2010 May;8(5):665-76. doi: 10.1158/1541-7786.MCR-09-0351. Epub 2010 Apr 20. Mol Cancer Res. 2010. PMID: 20407016

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