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Apoptosis signal-regulating kinase 1 inhibition attenuates cardiac hypertrophy and cardiorenal fibrosis induced by uremic toxins: Implications for cardiorenal syndrome.
Savira F, Cao L, Wang I, Yang W, Huang K, Hua Y, Jucker BM, Willette RN, Huang L, Krum H, Li Z, Fu Q, Wang BH. Savira F, et al. Among authors: huang l, huang k. PLoS One. 2017 Nov 6;12(11):e0187459. doi: 10.1371/journal.pone.0187459. eCollection 2017. PLoS One. 2017. PMID: 29107962 Free PMC article.
Vasculopathy in the setting of cardiorenal syndrome: roles of protein-bound uremic toxins.
Guo J, Lu L, Hua Y, Huang K, Wang I, Huang L, Fu Q, Chen A, Chan P, Fan H, Liu ZM, Wang BH. Guo J, et al. Among authors: huang l, huang k. Am J Physiol Heart Circ Physiol. 2017 Jul 1;313(1):H1-H13. doi: 10.1152/ajpheart.00787.2016. Epub 2017 Apr 14. Am J Physiol Heart Circ Physiol. 2017. PMID: 28411233 Free article. Review.
Sphingolipid imbalance and inflammatory effects induced by uremic toxins in heart and kidney cells are reversed by dihydroceramide desaturase 1 inhibition.
Savira F, Magaye R, Scullino CV, Flynn BL, Pitson SM, Anderson D, Creek DJ, Hua Y, Xiong X, Huang L, Liew D, Reid C, Kaye D, Kompa AR, Wang BH. Savira F, et al. Among authors: huang l. Toxicol Lett. 2021 Oct 10;350:133-142. doi: 10.1016/j.toxlet.2021.07.012. Epub 2021 Jul 22. Toxicol Lett. 2021. PMID: 34303789
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