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113 results

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Blockade of 20-hydroxyeicosatetraenoic acid receptor lowers blood pressure and alters vascular function in mice with smooth muscle-specific overexpression of CYP4A12-20-HETE synthase.
Agostinucci K, Hutcheson R, Hossain S, Pascale JV, Villegas E, Zhang F, Adebesin AM, Falck JR, Gupte S, Garcia V, Schwartzman ML. Agostinucci K, et al. Among authors: hutcheson r. J Hypertens. 2022 Mar 1;40(3):498-511. doi: 10.1097/HJH.0000000000003038. J Hypertens. 2022. PMID: 35081581 Free PMC article.
Elevated 20-HETE impairs coronary collateral growth in metabolic syndrome via endothelial dysfunction.
Joseph G, Soler A, Hutcheson R, Hunter I, Bradford C, Hutcheson B, Gotlinger KH, Jiang H, Falck JR, Proctor S, Schwartzman ML, Rocic P. Joseph G, et al. Among authors: hutcheson r, hutcheson b. Am J Physiol Heart Circ Physiol. 2017 Mar 1;312(3):H528-H540. doi: 10.1152/ajpheart.00561.2016. Epub 2016 Dec 23. Am J Physiol Heart Circ Physiol. 2017. PMID: 28011587 Free PMC article.
Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation.
Soler A, Hunter I, Joseph G, Hutcheson R, Hutcheson B, Yang J, Zhang FF, Joshi SR, Bradford C, Gotlinger KH, Maniyar R, Falck JR, Proctor S, Schwartzman ML, Gupte SA, Rocic P. Soler A, et al. Among authors: hutcheson r, hutcheson b. J Mol Cell Cardiol. 2018 Apr;117:88-99. doi: 10.1016/j.yjmcc.2018.02.005. Epub 2018 Feb 8. J Mol Cell Cardiol. 2018. PMID: 29428638 Free PMC article.
Corrigendum to "Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation" [J. Mol. Cell. Cardiol. 117 (2018) 88-99].
Soler A, Hunter I, Joseph G, Hutcheson R, Hutcheson B, Yang J, Zhang FF, Joshi SR, Bradford C, Gotlinger KH, Maniyar R, Falck JR, Proctor S, Schwartzman ML, Gupte SA, Rocic P. Soler A, et al. Among authors: hutcheson r, hutcheson b. J Mol Cell Cardiol. 2018 Aug;121:308. doi: 10.1016/j.yjmcc.2018.03.015. Epub 2018 Apr 4. J Mol Cell Cardiol. 2018. PMID: 29625729 No abstract available.
Impaired coronary collateral growth in the metabolic syndrome is in part mediated by matrix metalloproteinase 12-dependent production of endostatin and angiostatin.
Dodd T, Wiggins L, Hutcheson R, Smith E, Musiyenko A, Hysell B, Russell JC, Rocic P. Dodd T, et al. Among authors: hutcheson r. Arterioscler Thromb Vasc Biol. 2013 Jun;33(6):1339-49. doi: 10.1161/ATVBAHA.113.301533. Epub 2013 Apr 18. Arterioscler Thromb Vasc Biol. 2013. PMID: 23599440 Free PMC article.
miR-21-mediated decreased neutrophil apoptosis is a determinant of impaired coronary collateral growth in metabolic syndrome.
Hutcheson R, Terry R, Hutcheson B, Jadhav R, Chaplin J, Smith E, Barrington R, Proctor SD, Rocic P. Hutcheson R, et al. Among authors: hutcheson b. Am J Physiol Heart Circ Physiol. 2015 Jun 1;308(11):H1323-35. doi: 10.1152/ajpheart.00654.2014. Epub 2015 Apr 3. Am J Physiol Heart Circ Physiol. 2015. PMID: 25840830 Free PMC article.
Mechanisms of Comorbidities Associated With the Metabolic Syndrome: Insights from the JCR:LA-cp Corpulent Rat Strain.
Diane A, Pierce WD, Kelly SE, Sokolik S, Borthwick F, Jacome-Sosa M, Mangat R, Pradillo JM, Allan SM, Ruth MR, Field CJ, Hutcheson R, Rocic P, Russell JC, Vine DF, Proctor SD. Diane A, et al. Among authors: hutcheson r. Front Nutr. 2016 Oct 10;3:44. doi: 10.3389/fnut.2016.00044. eCollection 2016. Front Nutr. 2016. PMID: 27777929 Free PMC article. Review.
113 results